1-hexadecyl-2-acetyl-glycero-3-phosphocholine has been researched along with Bacterial Pneumonia in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (50.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Albertí, S; Barbier, M; Blázquez, J; Damron, FH; Goldberg, JB; Gomila, R; Martínez-Ramos, I; Owings, JP | 1 |
de Lima, CD; de Oliveira, AV; Machado, GB; Plotkowski, MC; Saliba, AM; Suassuna, JH | 1 |
de Beer, R; de Boer, OJ; de Vos, AF; Florquin, S; Meijers, JC; van der Poll, T; van Zoelen, MA | 1 |
Draing, C; Golenbock, DT; Haslinger, I; Knapp, S; Leendertse, M; van der Poll, T; von Aulock, S | 1 |
4 other study(ies) available for 1-hexadecyl-2-acetyl-glycero-3-phosphocholine and Bacterial Pneumonia
Article | Year |
---|---|
Lysine trimethylation of EF-Tu mimics platelet-activating factor to initiate Pseudomonas aeruginosa pneumonia.
Topics: Adhesins, Bacterial; Animals; Bacterial Adhesion; Female; Lysine; Male; Methylation; Methyltransferases; Mice; Mice, Inbred BALB C; Peptide Elongation Factor Tu; Platelet Activating Factor; Pneumonia, Bacterial; Protein Processing, Post-Translational; Pseudomonas aeruginosa; Virulence Factors | 2013 |
Pseudomonas aeruginosa toxin ExoU induces a PAF-dependent impairment of alveolar fibrin turnover secondary to enhanced activation of coagulation and increased expression of plasminogen activator inhibitor-1 in the course of mice pneumosepsis.
Topics: Animals; Bacterial Proteins; Blood Coagulation; Bronchoalveolar Lavage Fluid; Cell Line, Tumor; Disease Models, Animal; Epithelial Cells; Female; Fibrin; Humans; Immunohistochemistry; Macrophages; Mice; Mutation; Plasminogen Activator Inhibitor 1; Platelet Activating Factor; Pneumonia, Bacterial; Pseudomonas aeruginosa; Pseudomonas Infections; Pulmonary Alveoli; Respiratory Mucosa; RNA, Messenger; Sepsis; Time Factors; Up-Regulation | 2011 |
Platelet-activating factor receptor contributes to host defense against Pseudomonas aeruginosa pneumonia but is not essential for the accompanying inflammatory and procoagulant response.
Topics: Animals; Inflammation Mediators; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Platelet Activating Factor; Platelet Membrane Glycoproteins; Pneumonia, Bacterial; Pseudomonas Infections; Receptors, G-Protein-Coupled | 2008 |
Lipoteichoic acid-induced lung inflammation depends on TLR2 and the concerted action of TLR4 and the platelet-activating factor receptor.
Topics: Acute Disease; Animals; Cell Line; Female; Humans; Inflammation Mediators; Lipopolysaccharide Receptors; Lipopolysaccharides; Lung; Mice; Mice, Inbred C57BL; Mice, Knockout; Platelet Activating Factor; Platelet Membrane Glycoproteins; Pneumonia, Bacterial; Pulmonary Alveoli; Receptors, G-Protein-Coupled; Teichoic Acids; Toll-Like Receptor 2; Toll-Like Receptor 4 | 2008 |