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1-anilino-8-naphthalenesulfonate and Multiple Sclerosis

1-anilino-8-naphthalenesulfonate has been researched along with Multiple Sclerosis in 8 studies

1-anilino-8-naphthalenesulfonate: RN given refers to parent cpd
8-anilinonaphthalene-1-sulfonic acid : A naphthalenesulfonic acid that is naphthalene-1-sulfonic acid substituted by a phenylamino group at position 8.

Multiple Sclerosis: An autoimmune disorder mainly affecting young adults and characterized by destruction of myelin in the central nervous system. Pathologic findings include multiple sharply demarcated areas of demyelination throughout the white matter of the central nervous system. Clinical manifestations include visual loss, extra-ocular movement disorders, paresthesias, loss of sensation, weakness, dysarthria, spasticity, ataxia, and bladder dysfunction. The usual pattern is one of recurrent attacks followed by partial recovery (see MULTIPLE SCLEROSIS, RELAPSING-REMITTING), but acute fulminating and chronic progressive forms (see MULTIPLE SCLEROSIS, CHRONIC PROGRESSIVE) also occur. (Adams et al., Principles of Neurology, 6th ed, p903)

Research

Studies (8)

TimeframeStudies, this research(%)All Research%
pre-19907 (87.50)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's1 (12.50)2.80

Authors

AuthorsStudies
Capone, F1
De Vincentis, A1
Ferraro, E1
Rossi, M1
Motolese, F1
Picardi, A1
Giannetti, B1
Di Battista, G1
Vespasiani-Gentilucci, U1
Di Lazzaro, V1
VOGEL, FS1
LESNY, I2
POLACEK, L2
EICHLER, V1
Hirsch, HE2
Neu, I1
Woelk, H1
Duquette, P1
Parks, ME1
Kluge, H1
Wieczorek, V1

Other Studies

8 other studies available for 1-anilino-8-naphthalenesulfonate and Multiple Sclerosis

ArticleYear
The PNPLA3 rs738409 variant can increase the risk of liver toxicity in multiple sclerosis patients treated with beta-interferon.
    Clinical neurology and neurosurgery, 2020, Volume: 197

    Topics: Adult; Chemical and Drug Induced Liver Injury; Female; Genetic Predisposition to Disease; Humans; In

2020
Demyelinization induced in living rabbits by means of a lipolytic enzyme preparation.
    The Journal of experimental medicine, 1951, Apr-01, Volume: 93, Issue:4

    Topics: Animals; Brain; Central Nervous System; Enzymes; Guinea Pigs; Lipase; Multiple Sclerosis; Myelin She

1951
[Fluctuations in the lipolytic power of the serum of patients with multiple sclerosis].
    Acta neurologica et psychiatrica Belgica, 1951, Volume: 51, Issue:9

    Topics: Humans; Lipase; Lipolysis; Multiple Sclerosis

1951
[Variation of blood lipase level in disseminated sclerosis].
    Biologicke listy, 1951, Volume: 32, Issue:3

    Topics: Humans; Lipase; Multiple Sclerosis

1951
Pathogenesis of demyelination in multiple sclerosis: contributions of microanalysis.
    Progress in clinical and biological research, 1980, Volume: 39

    Topics: Astrocytes; Body Water; Carboxypeptidase B; Carboxypeptidases; Demyelinating Diseases; DNA; Endopept

1980
Investigations of the lipid metabolism of the white matter in multiple sclerosis: changes in glycero-phosphatides and lipid-splitting enzymes.
    Neurochemical research, 1982, Volume: 7, Issue:6

    Topics: Adult; Brain; Fatty Acids; Humans; Lipase; Lipids; Male; Multiple Sclerosis; Phosphatidylglycerols;

1982
The quantitative histochemistry of multiple sclerosis plaques: acid proteinase and other acid hydrolases.
    Journal of neurochemistry, 1976, Volume: 26, Issue:3

    Topics: Acid Phosphatase; Brain; DNA; Female; Galactosidases; Glucosephosphate Dehydrogenase; Glucuronidase;

1976
[Lipase activities in the cerebrospinal fluid].
    Psychiatrie, Neurologie, und medizinische Psychologie, 1968, Volume: 20, Issue:9

    Topics: Adult; Atrophy; Brain Diseases; Brain Neoplasms; Central Nervous System Diseases; Clinical Enzyme Te

1968