1-aminoindan-1,5-dicarboxylic acid has been researched along with Ischemic Attack, Transient in 3 studies
1-aminoindan-1,5-dicarboxylic acid: structure given in first source
Ischemic Attack, Transient: Brief reversible episodes of focal, nonconvulsive ischemic dysfunction of the brain having a duration of less than 24 hours, and usually less than one hour, caused by transient thrombotic or embolic blood vessel occlusion or stenosis. Events may be classified by arterial distribution, temporal pattern, or etiology (e.g., embolic vs. thrombotic). (From Adams et al., Principles of Neurology, 6th ed, pp814-6)
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 2 (66.67) | 18.2507 |
2000's | 1 (33.33) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Meli, E | 3 |
Picca, R | 1 |
Attucci, S | 1 |
Cozzi, A | 3 |
Peruginelli, F | 3 |
Moroni, F | 3 |
Pellegrini-Giampietro, DE | 3 |
Albani-Torregrossa, S | 1 |
Pellicciari, R | 2 |
Leonardi, P | 1 |
3 other studies available for 1-aminoindan-1,5-dicarboxylic acid and Ischemic Attack, Transient
Article | Year |
---|---|
Activation of mGlu1 but not mGlu5 metabotropic glutamate receptors contributes to postischemic neuronal injury in vitro and in vivo.
Topics: Animals; Brain Ischemia; Bridged Bicyclo Compounds; Cell Line; Cricetinae; Excitatory Amino Acid Ago | 2002 |
Protection with metabotropic glutamate 1 receptor antagonists in models of ischemic neuronal death: time-course and mechanisms.
Topics: Animals; Bridged Bicyclo Compounds; Cell Hypoxia; Cells, Cultured; Cerebral Cortex; Coculture Techni | 1999 |
1-Aminoindan-1,5-dicarboxylic acid and (S)-(+)-2-(3'-carboxybicyclo[1.1.1] pentyl)-glycine, two mGlu1 receptor-preferring antagonists, reduce neuronal death in in vitro and in vivo models of cerebral ischaemia.
Topics: Animals; Animals, Newborn; Astrocytes; Benzoates; Bridged Bicyclo Compounds; Cell Death; Cells, Cult | 1999 |