1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester has been researched along with Skin Neoplasms in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (66.67) | 29.6817 |
2010's | 1 (33.33) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Dutta, RK; Ishaq, M; Khan, MA; Majumdar, S; Sharma, G; Sharma, K | 1 |
Baugher, KM; Bernier, M; Camilli, TC; Earley, R; Elias, EG; Fiori, JL; Frank, BP; French, AD; Hasskamp, JH; Hoek, KS; Indig, FE; O'Connell, MP; Taub, DD; Weeraratna, AT | 1 |
Chignell, CF; He, YY; Huang, JL | 1 |
3 other study(ies) available for 1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester and Skin Neoplasms
Article | Year |
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Pentoxifylline triggers autophagy via ER stress response that interferes with Pentoxifylline induced apoptosis in human melanoma cells.
Topics: Antineoplastic Agents; Apoptosis; Autophagy; Autophagy-Related Protein 5; Calcium; Calcium Chelating Agents; Cell Line, Tumor; Drug Resistance, Neoplasm; Egtazic Acid; Endoplasmic Reticulum Chaperone BiP; Endoplasmic Reticulum Stress; Humans; Intracellular Space; Melanoma; Microtubule-Associated Proteins; Pentoxifylline; Phosphodiesterase Inhibitors; Signal Transduction; Skin Neoplasms; TOR Serine-Threonine Kinases; Transcription Factor CHOP | 2016 |
Wnt5A activates the calpain-mediated cleavage of filamin A.
Topics: Calpain; Cell Line, Tumor; Cell Movement; Chelating Agents; Contractile Proteins; Cytoskeleton; Egtazic Acid; Filamins; Gene Expression Regulation, Neoplastic; Humans; Melanoma; Microfilament Proteins; Proto-Oncogene Proteins; Receptor Tyrosine Kinase-like Orphan Receptors; Receptors, Cell Surface; RNA, Small Interfering; Skin Neoplasms; Wnt Proteins; Wnt-5a Protein | 2009 |
Delayed and sustained activation of extracellular signal-regulated kinase in human keratinocytes by UVA: implications in carcinogenesis.
Topics: Animals; Apoptosis; Blotting, Western; Calcium; Carbazoles; Caspase 3; Caspases; Cell Line; Cell Line, Tumor; Cell Membrane; Cell Survival; Cell Transformation, Neoplastic; Chelating Agents; COS Cells; Cytosol; Dose-Response Relationship, Radiation; Egtazic Acid; Enzyme Activation; Enzyme Inhibitors; ErbB Receptors; Estrenes; Flow Cytometry; Genes, Dominant; Golgi Apparatus; Humans; Indoles; Keratinocytes; Mitogen-Activated Protein Kinase 3; Models, Biological; Neoplasms; Plasmids; Protein Kinase C; Pyrrolidinones; ras Proteins; Skin Neoplasms; Subcellular Fractions; Tetradecanoylphorbol Acetate; Time Factors; Transfection; Ultraviolet Rays | 2004 |