Compounds > 1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester

1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester and Granulocytic Leukemia, Chronic

1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester has been researched along with Granulocytic Leukemia, Chronic in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	2 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Boffo, S; Caparrotti, G; Ciarcia, R; d'Angelo, D; Damiano, S; Florio, S; Giordano, A; Montagnaro, S; Morales, F; Pagnini, U; Rizzolio, F; Ruocco, A	1
Heise, N; Henke, G; Huber, SM; Koka, S; Lang, F; Misovic, M; Palme, D; Rudner, J; Salih, HR	1

Other Studies

2 other study(ies) available for 1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester and Granulocytic Leukemia, Chronic

Article	Year
Combined effects of PI3K and SRC kinase inhibitors with imatinib on intracellular calcium levels, autophagy, and apoptosis in CML-PBL cells. Cell cycle (Georgetown, Tex.), 2013, Sep-01, Volume: 12, Issue:17 Topics: Apoptosis; Autophagy; Benzamides; Calcium; Calcium Signaling; Cell Line, Tumor; Chromones; Drug Synergism; Egtazic Acid; Humans; Imatinib Mesylate; Inositol Phosphates; Intracellular Space; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Morpholines; Phosphatidylinositol 3-Kinase; Phosphoinositide-3 Kinase Inhibitors; Piperazines; Protein Kinase Inhibitors; Pyrazoles; Pyrimidines; src-Family Kinases; Thapsigargin	2013
Non-selective cation channel-mediated Ca2+-entry and activation of Ca2+/calmodulin-dependent kinase II contribute to G2/M cell cycle arrest and survival of irradiated leukemia cells. Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 2010, Volume: 26, Issue:4-5 Topics: Calcium; Calcium Channels; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Cell Division; Chelating Agents; Egtazic Acid; Fura-2; G2 Phase; Humans; K562 Cells; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Patch-Clamp Techniques; Protein Kinase Inhibitors; Radiation Tolerance; Radiation, Ionizing; TRPV Cation Channels	2010

Article

Year

Combined effects of PI3K and SRC kinase inhibitors with imatinib on intracellular calcium levels, autophagy, and apoptosis in CML-PBL cells.

Cell cycle (Georgetown, Tex.), 2013, Sep-01, Volume: 12, Issue:17

Topics: Apoptosis; Autophagy; Benzamides; Calcium; Calcium Signaling; Cell Line, Tumor; Chromones; Drug Synergism; Egtazic Acid; Humans; Imatinib Mesylate; Inositol Phosphates; Intracellular Space; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Morpholines; Phosphatidylinositol 3-Kinase; Phosphoinositide-3 Kinase Inhibitors; Piperazines; Protein Kinase Inhibitors; Pyrazoles; Pyrimidines; src-Family Kinases; Thapsigargin

2013

Non-selective cation channel-mediated Ca2+-entry and activation of Ca2+/calmodulin-dependent kinase II contribute to G2/M cell cycle arrest and survival of irradiated leukemia cells.

Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 2010, Volume: 26, Issue:4-5

Topics: Calcium; Calcium Channels; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Cell Division; Chelating Agents; Egtazic Acid; Fura-2; G2 Phase; Humans; K562 Cells; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Patch-Clamp Techniques; Protein Kinase Inhibitors; Radiation Tolerance; Radiation, Ionizing; TRPV Cation Channels

2010