1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester and Breast Cancer

1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester has been researched along with Breast Cancer in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (33.33)18.2507
2000's2 (33.33)29.6817
2010's2 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chang, HT; Chou, CT; Jan, CR; Kuo, DH; Liang, WZ; Shieh, P1
Ghosh, S; Hazra, B; Kumar, A; Kumar, B; Mishra, KP; Pandey, BN1
Glover-Collins, K; Thompson, ME1
Hagen, K; Kong, AN; Mandlekar, S; Roninson, IB; Shtil, AA; Tan, TH; Walter, RJ; Yu, R1
Gist, ID; Kiang, JG; Tsokos, GC1
Boothman, DA; Dubyak, GR; Nieminen, AL; Pink, JJ; Tagliarino, C1

Other Studies

6 other study(ies) available for 1,2-bis(2-aminophenoxy)ethane n,n,n',n'-tetraacetic acid acetoxymethyl ester and Breast Cancer

ArticleYear
The Mechanism of Ca(2+) Movement in the Involvement of Baicalein-Induced Cytotoxicity in ZR-75-1 Human Breast Cancer Cells.
    Journal of natural products, 2015, Jul-24, Volume: 78, Issue:7

    Topics: Acetylcysteine; Animals; Apoptosis; Boron Compounds; Breast Neoplasms; Calcium; Caspase 3; Caspase 9; Cytosol; Egtazic Acid; Endoplasmic Reticulum; Female; Flavanones; Humans; Indoles; Maleimides; Molecular Structure; Reactive Oxygen Species; Thapsigargin; Type C Phospholipases

2015
Diospyrin derivative, an anticancer quinonoid, regulates apoptosis at endoplasmic reticulum as well as mitochondria by modulating cytosolic calcium in human breast carcinoma cells.
    Biochemical and biophysical research communications, 2012, Jan-13, Volume: 417, Issue:2

    Topics: Acetylcysteine; Antineoplastic Agents; Apoptosis; Breast Neoplasms; Calcium; Calcium Channels; Carcinoma; Cell Line, Tumor; Chelating Agents; Cytosol; Egtazic Acid; Endoplasmic Reticulum; Estrenes; Female; Humans; Mitochondria; Naphthoquinones; Oxidation-Reduction; Phosphodiesterase Inhibitors; Pyrrolidinones; Ruthenium Red; Type C Phospholipases

2012
Nuclear export of BRCA1 occurs during early S phase and is calcium-dependent.
    Cellular signalling, 2008, Volume: 20, Issue:5

    Topics: Active Transport, Cell Nucleus; Apoptosis Regulatory Proteins; BRCA1 Protein; BRCA2 Protein; Breast; Breast Neoplasms; Calcimycin; Calcium; Calcium Signaling; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Cell Line, Tumor; Egtazic Acid; Epithelial Cells; Fatty Acids, Unsaturated; Female; Humans; S Phase; Signal Transduction

2008
Differential regulation of mitogen-activated protein kinases by microtubule-binding agents in human breast cancer cells.
    Oncogene, 1999, Jan-14, Volume: 18, Issue:2

    Topics: Apoptosis; Breast Neoplasms; Calcium; Calcium-Calmodulin-Dependent Protein Kinases; Cell Cycle; Colchicine; Egtazic Acid; Enzyme Activation; Humans; Microtubules; Nocodazole; Paclitaxel; Podophyllotoxin; Tumor Cells, Cultured; Vincristine

1999
Biochemical requirements for the expression of heat shock protein 72 kda in human breast cancer MCF-7 cells.
    Molecular and cellular biochemistry, 1999, Volume: 199, Issue:1-2

    Topics: Breast Neoplasms; Calcium; Calcium Signaling; Chelating Agents; Cyclic AMP-Dependent Protein Kinases; Cytosol; Egtazic Acid; Enzyme Inhibitors; Female; Heat-Shock Proteins; Hot Temperature; HSP72 Heat-Shock Proteins; HSP90 Heat-Shock Proteins; Humans; Isoquinolines; Protein Kinase C; Receptors, Estrogen; Sulfonamides; Tumor Cells, Cultured

1999
Calcium is a key signaling molecule in beta-lapachone-mediated cell death.
    The Journal of biological chemistry, 2001, Jun-01, Volume: 276, Issue:22

    Topics: 4-Nitroquinoline-1-oxide; Adenosine Triphosphate; Apoptosis; Blotting, Western; Breast Neoplasms; Calcium; Cell Death; Cell Division; Chelating Agents; Cytosol; DNA Fragmentation; Dose-Response Relationship, Drug; Egtazic Acid; Endoplasmic Reticulum; Female; Flow Cytometry; Humans; In Situ Nick-End Labeling; Male; Membrane Potentials; Microscopy, Confocal; Mitochondria; Models, Biological; Naphthoquinones; Prostatic Neoplasms; Quinolones; Signal Transduction; Time Factors; Tumor Cells, Cultured

2001