1-(2-(3-(4-methoxyphenyl)propoxy)-4-methoxyphenylethyl)-1h-imidazole and Prostatic Neoplasms

1-(2-(3-(4-methoxyphenyl)propoxy)-4-methoxyphenylethyl)-1h-imidazole has been researched along with Prostatic Neoplasms in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (50.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Selvaraj, S; Singh, BB; Sukumaran, P; Sun, Y	1
Bonnal, JL; Dewailly, E; Lemonnier, L; Mauroy, B; Prevarskaya, N; Roudbaraki, M; Shuba, Y; Skryma, R; Slomianny, C; Sydorenko, V; Thebault, S	1

Other Studies

2 other study(ies) available for 1-(2-(3-(4-methoxyphenyl)propoxy)-4-methoxyphenylethyl)-1h-imidazole and Prostatic Neoplasms

Article	Year
Resveratrol activates autophagic cell death in prostate cancer cells via downregulation of STIM1 and the mTOR pathway. Molecular carcinogenesis, 2016, Volume: 55, Issue:5 Topics: Antineoplastic Agents, Phytogenic; Autophagy; Calcium; Cell Line, Tumor; Cell Proliferation; Dose-Response Relationship, Drug; Down-Regulation; Endoplasmic Reticulum Stress; Gene Expression Regulation, Neoplastic; Humans; Imidazoles; Male; Membrane Proteins; Neoplasm Proteins; Prostatic Neoplasms; Resveratrol; Signal Transduction; Stilbenes; Stromal Interaction Molecule 1; TOR Serine-Threonine Kinases	2016
Alpha1-adrenergic receptors activate Ca(2+)-permeable cationic channels in prostate cancer epithelial cells. The Journal of clinical investigation, 2003, Volume: 111, Issue:11 Topics: Adrenergic alpha-1 Receptor Antagonists; Adrenergic alpha-Antagonists; Blotting, Western; Boron Compounds; Calcium; Calcium Channel Blockers; Cations; Cell Division; Diglycerides; Dioxanes; Electrophysiology; Epithelial Cells; Humans; Imidazoles; Male; Microscopy, Fluorescence; Prazosin; Prostatic Neoplasms; Receptors, Adrenergic, alpha-1; Reverse Transcriptase Polymerase Chain Reaction; Time Factors; Tumor Cells, Cultured	2003

Article

Year

Resveratrol activates autophagic cell death in prostate cancer cells via downregulation of STIM1 and the mTOR pathway.

Molecular carcinogenesis, 2016, Volume: 55, Issue:5

Topics: Antineoplastic Agents, Phytogenic; Autophagy; Calcium; Cell Line, Tumor; Cell Proliferation; Dose-Response Relationship, Drug; Down-Regulation; Endoplasmic Reticulum Stress; Gene Expression Regulation, Neoplastic; Humans; Imidazoles; Male; Membrane Proteins; Neoplasm Proteins; Prostatic Neoplasms; Resveratrol; Signal Transduction; Stilbenes; Stromal Interaction Molecule 1; TOR Serine-Threonine Kinases

2016

Alpha1-adrenergic receptors activate Ca(2+)-permeable cationic channels in prostate cancer epithelial cells.

The Journal of clinical investigation, 2003, Volume: 111, Issue:11

Topics: Adrenergic alpha-1 Receptor Antagonists; Adrenergic alpha-Antagonists; Blotting, Western; Boron Compounds; Calcium; Calcium Channel Blockers; Cations; Cell Division; Diglycerides; Dioxanes; Electrophysiology; Epithelial Cells; Humans; Imidazoles; Male; Microscopy, Fluorescence; Prazosin; Prostatic Neoplasms; Receptors, Adrenergic, alpha-1; Reverse Transcriptase Polymerase Chain Reaction; Time Factors; Tumor Cells, Cultured

2003