1-(2-(3-(4-methoxyphenyl)propoxy)-4-methoxyphenylethyl)-1h-imidazole has been researched along with Necrosis in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| He, Y; Mei, M; Shen, B; Wu, X; Zhao, H; Zhou, S | 1 |
| Chun, JN; Jeon, JH; Kim, BJ; Kim, IS; Kim, SH; Kim, SY; Lee, CR; Lee, SW; Park, EJ; Park, JK; So, I | 1 |
| Chatham, JC; Marchase, RB; Shan, D | 1 |
3 other study(ies) available for 1-(2-(3-(4-methoxyphenyl)propoxy)-4-methoxyphenylethyl)-1h-imidazole and Necrosis
| Article | Year |
|---|---|
TRPC6 May Protect Renal Ischemia-Reperfusion Injury Through Inhibiting Necroptosis of Renal Tubular Epithelial Cells.
Topics: Apoptosis; Benzylamines; Blotting, Western; Cell Line; Cell Shape; Down-Regulation; Epithelial Cells; Fluorescent Antibody Technique; Humans; Imidazoles; Ischemia; Kidney Tubules; Necrosis; Oxygen; Protective Agents; Reperfusion Injury; RNA, Small Interfering; Sulfonamides; TRPC Cation Channels; TRPC6 Cation Channel; Up-Regulation | 2016 |
SK&F 96365 induces apoptosis and autophagy by inhibiting Akt-mTOR signaling in A7r5 cells.
Topics: Apoptosis; Autophagy; Blotting, Western; Calcium; Calcium Channel Blockers; Cell Proliferation; Cells, Cultured; Flow Cytometry; Humans; Imidazoles; Muscle, Smooth, Vascular; Necrosis; Proto-Oncogene Proteins c-akt; Signal Transduction; TOR Serine-Threonine Kinases | 2011 |
Overexpression of TRPC3 increases apoptosis but not necrosis in response to ischemia-reperfusion in adult mouse cardiomyocytes.
Topics: Acrylates; Animals; Apoptosis; Calcium; Calcium Channel Blockers; Calcium Channels, L-Type; Calcium Signaling; Calpain; Carrier Proteins; Cell Survival; Cysteine Proteinase Inhibitors; Imidazoles; Male; Mice; Mice, Transgenic; Microfilament Proteins; Myocardial Reperfusion Injury; Myocytes, Cardiac; Necrosis; Time Factors; TRPC Cation Channels; Tumor Necrosis Factor-alpha; Up-Regulation; Verapamil | 2008 |