Page last updated: 2024-08-16

w 7 and 4-bromophenacyl bromide

w 7 has been researched along with 4-bromophenacyl bromide in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19902 (66.67)18.7374
1990's1 (33.33)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Gleich, GJ; Thomas, LL; Zheutlin, LM1
Nakagawa, Y; Waku, K1
Geertsma, MF; Nibbering, PH; Van Den Barselaar, MT; Van Furth, R; Zheng, L; Zomerdijk, TP1

Other Studies

3 other study(ies) available for w 7 and 4-bromophenacyl bromide

ArticleYear
Pharmacological control of human basophil histamine release stimulated by eosinophil granule major basic protein.
    Immunology, 1989, Volume: 66, Issue:4

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; Acetophenones; Basophils; Blood Proteins; Calcium; Eosinophil Granule Proteins; Eosinophils; Histamine Release; Humans; Isoquinolines; Pertussis Toxin; Phospholipases A; Phospholipases A2; Piperazines; Protein Kinase C; Ribonucleases; Sulfonamides; Theophylline; Virulence Factors, Bordetella

1989
Selective inhibition of free arachidonic acid production in activated alveolar macrophages by calmodulin antagonists.
    Biochemical and biophysical research communications, 1988, Oct-31, Volume: 156, Issue:2

    Topics: Acetophenones; Animals; Arachidonic Acid; Arachidonic Acids; Calmodulin; Chlorpromazine; Chromatography, High Pressure Liquid; Cytochalasin B; Linoleic Acid; Linoleic Acids; Macrophage Activation; Macrophages; Oleic Acid; Oleic Acids; Opsonin Proteins; Phosphatidylcholines; Phosphatidylinositols; Phospholipases A; Pulmonary Alveoli; Quinacrine; Rabbits; Sulfonamides; Trifluoperazine; Zymosan

1988
Arachidonic acid, but not its metabolites, is essential for FcgammaR-stimulated intracellular killing of Staphylococcus aureus by human monocytes.
    Immunology, 1999, Volume: 96, Issue:1

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 4,5-Dihydro-1-(3-(trifluoromethyl)phenyl)-1H-pyrazol-3-amine; Acetophenones; Arachidonic Acid; Calcium-Calmodulin-Dependent Protein Kinases; Cyclic AMP-Dependent Protein Kinases; Cyclooxygenase Inhibitors; Dose-Response Relationship, Drug; Enzyme Inhibitors; Genistein; Humans; Immunoglobulin G; Indomethacin; Lipoxygenase Inhibitors; Monocytes; Oxygen; Phagocytosis; Phospholipases A; Phospholipases A2; Protein Kinase C; Protein-Tyrosine Kinases; Receptors, IgG; Staphylococcus aureus; Sulfonamides

1999