toxoflavin has been researched along with icg 001 in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Akan, H; Beyer, C; Dees, C; Distler, A; Distler, JH; Distler, O; Gelse, K; Lin, NY; Mallano, T; Palumbo-Zerr, K; Reichert, H; Schett, G; Schramm, A; Varga, J | 1 |
Ayadi, M; Bouygues, A; Chouaib, S; Ferrand, N; Larsen, AK; Muchardt, C; Ouaret, D; Sabbah, M; Thiery, JP | 1 |
Almonte, V; Chinnasamy, P; Egaña-Gorroño, L; Gross, JN; Guo, L; Jayakumar, S; Parikh, D; Riascos-Bernal, DF; Sibinga, NES | 1 |
3 other study(ies) available for toxoflavin and icg 001
Article | Year |
---|---|
Blockade of canonical Wnt signalling ameliorates experimental dermal fibrosis.
Topics: Animals; Bridged Bicyclo Compounds, Heterocyclic; Disease Models, Animal; Fibrosis; Mice; Mice, Inbred DBA; Pyrimidinones; Scleroderma, Systemic; Signal Transduction; Skin; Skin Diseases; Treatment Outcome; Triazines; Wnt Signaling Pathway | 2013 |
Chronic chemotherapeutic stress promotes evolution of stemness and WNT/beta-catenin signaling in colorectal cancer cells: implications for clinical use of WNT-signaling inhibitors.
Topics: Antineoplastic Agents; Blotting, Western; Bridged Bicyclo Compounds, Heterocyclic; Camptothecin; Cell Line, Tumor; Cell Survival; Colorectal Neoplasms; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; Fluorouracil; Gene Expression Regulation, Neoplastic; HCT116 Cells; HT29 Cells; Humans; Hyaluronan Receptors; Irinotecan; Neoplastic Stem Cells; Organoplatinum Compounds; Oxaliplatin; Protein Isoforms; Pyrimidinones; Reverse Transcriptase Polymerase Chain Reaction; Triazines; Wnt Signaling Pathway | 2015 |
Inhibition of Smooth Muscle β-Catenin Hinders Neointima Formation After Vascular Injury.
Topics: Animals; Apoptosis; beta Catenin; Bridged Bicyclo Compounds, Heterocyclic; Carotid Arteries; Carotid Artery Injuries; Cell Movement; Cell Proliferation; Cells, Cultured; Disease Models, Animal; Dose-Response Relationship, Drug; Gene Expression Regulation; Genotype; Humans; Male; Mice, Knockout; Muscle, Smooth, Vascular; Myocytes, Smooth Muscle; Neointima; Phenotype; Pyrimidinones; Signal Transduction; Time Factors; Triazines; Vascular Remodeling | 2017 |