thalidomide has been researched along with cytochrome c-t in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (100.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Domschke, W; Gockel, HR; Heidemann, J; Kucharzik, T; Lügering, A; Lügering, N; Schmidt, M | 1 |
Du, GJ; Lin, HH; Wang, MW; Xu, QT | 1 |
2 other study(ies) available for thalidomide and cytochrome c-t
Article | Year |
---|---|
Thalidomide induces apoptosis in human monocytes by using a cytochrome c-dependent pathway.
Topics: Apoptosis; Apoptosis Regulatory Proteins; Caspase 9; Caspases; Cells, Cultured; Cytochromes c; Enzyme Activation; Fas Ligand Protein; fas Receptor; Humans; Membrane Glycoproteins; Mitochondria; Monocytes; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Protein Serine-Threonine Kinases; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; Receptors, Tumor Necrosis Factor; Signal Transduction; Thalidomide; TNF-Related Apoptosis-Inducing Ligand; Tumor Necrosis Factor-alpha | 2004 |
Thalidomide inhibits growth of tumors through COX-2 degradation independent of antiangiogenesis.
Topics: Angiogenesis Inhibitors; Animals; Antineoplastic Agents; Blotting, Western; Cell Line, Tumor; Cell Proliferation; Cyclooxygenase 2; Cytochromes c; Dose-Response Relationship, Drug; Female; Glutathione; HeLa Cells; HL-60 Cells; Humans; K562 Cells; Male; Mice; Mice, Inbred BALB C; Mice, Nude; Neovascularization, Pathologic; Proto-Oncogene Proteins c-bcl-2; Thalidomide; Tumor Necrosis Factor-alpha; Vascular Endothelial Growth Factor A; Xenograft Model Antitumor Assays | 2005 |