substance p has been researched along with phorbol-12,13-didecanoate in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 1 (33.33) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (33.33) | 29.6817 |
2010's | 1 (33.33) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Obie, JF; Putney, JW; Sugiya, H | 1 |
Gao, F; Garavito, RM; Sui, D; Wang, DH; Worden, RM | 1 |
Gao, F; Wang, DH | 1 |
3 other study(ies) available for substance p and phorbol-12,13-didecanoate
Article | Year |
---|---|
Two modes of regulation of the phospholipase C-linked substance-P receptor in rat parotid acinar cells.
Topics: Animals; Binding Sites; Calcium; Enzyme Activation; In Vitro Techniques; Inositol 1,4,5-Trisphosphate; Inositol Phosphates; Parotid Gland; Phorbol 12,13-Dibutyrate; Phorbol Esters; Protein Kinase C; Rats; Rats, Inbred Strains; Receptors, Neurokinin-1; Receptors, Neurotransmitter; Substance P; Tetradecanoylphorbol Acetate; Type C Phospholipases | 1988 |
Salt intake augments hypotensive effects of transient receptor potential vanilloid 4: functional significance and implication.
Topics: Animals; Blood Pressure; Calcitonin Gene-Related Peptide; Capsaicin; Disease Models, Animal; Dose-Response Relationship, Drug; Ganglia, Spinal; Hypertension; Hypotension; Kidney; Male; Mesenteric Arteries; Phorbol Esters; Rats; Rats, Wistar; Ruthenium Red; Salt Tolerance; Sodium Chloride, Dietary; Substance P; TRPV Cation Channels | 2009 |
Hypotension induced by activation of the transient receptor potential vanilloid 4 channels: role of Ca2+-activated K+ channels and sensory nerves.
Topics: Animals; Animals, Newborn; Apamin; Blood Pressure; Calcitonin Gene-Related Peptide; Capsaicin; Charybdotoxin; Drug Combinations; Drug Therapy, Combination; Endothelium, Vascular; Hypotension; Injections, Subcutaneous; Male; Mesenteric Arteries; Nerve Degeneration; Neurons, Afferent; Peptide Fragments; Phorbol Esters; Potassium Channels, Calcium-Activated; Rats; Rats, Wistar; Sensory System Agents; Substance P; TRPV Cation Channels; Vasodilator Agents | 2010 |