substance p has been researched along with carbon monoxide in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (25.00) | 18.7374 |
| 1990's | 1 (25.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Anggård, A; Lundberg, JM; Lundblad, L; Saria, A | 1 |
| Jessop, DS | 1 |
| Mallard, C; McGregor, H; Rees, S; Tolcos, M; Walker, D | 1 |
| McGregor, H; Rees, S; Tolcos, M; Walker, D | 1 |
1 review(s) available for substance p and carbon monoxide
| Article | Year |
|---|---|
Review: Central non-glucocorticoid inhibitors of the hypothalamo-pituitary-adrenal axis.
Topics: Adrenocorticotropic Hormone; Adrenomedullin; Annexin A1; Atrial Natriuretic Factor; beta-Endorphin; Carbon Monoxide; Endothelins; gamma-Aminobutyric Acid; Humans; Hypothalamo-Hypophyseal System; Leptin; Neurotransmitter Agents; Nitric Oxide; Oxytocin; Peptides; Pituitary-Adrenal System; Protein Precursors; Proteins; Somatostatin; Stress, Psychological; Substance P; Thyrotropin-Releasing Hormone | 1999 |
3 other study(ies) available for substance p and carbon monoxide
| Article | Year |
|---|---|
Inhibition of cigarette smoke-induced oedema in the nasal mucosa by capsaicin pretreatment and a substance P antagonist.
Topics: Animals; Blood Pressure; Capillary Permeability; Capsaicin; Carbon Monoxide; Edema; Histamine Antagonists; Male; Nasal Mucosa; Nicotine; Rats; Rats, Inbred Strains; Smoking; Substance P | 1984 |
Exposure to prenatal carbon monoxide and postnatal hyperthermia: short and long-term effects on neurochemicals and neuroglia in the developing brain.
Topics: Animals; Animals, Newborn; Body Weight; Brain; Brain Chemistry; Carbon Monoxide; Cerebral Cortex; Enkephalin, Methionine; Female; Glial Fibrillary Acidic Protein; Guinea Pigs; Hyperthermia, Induced; Immunohistochemistry; Inhalation Exposure; Maternal Exposure; Medulla Oblongata; Neuroglia; Organ Size; Pregnancy; Prenatal Exposure Delayed Effects; Serotonin; Substance P; Sudden Infant Death; Thalamus; Tyrosine 3-Monooxygenase | 2000 |
Chronic prenatal exposure to carbon monoxide results in a reduction in tyrosine hydroxylase-immunoreactivity and an increase in choline acetyltransferase-immunoreactivity in the fetal medulla: implications for Sudden Infant Death Syndrome.
Topics: Animals; Astrocytes; Blotting, Western; Carbon Monoxide; Choline O-Acetyltransferase; Enkephalin, Methionine; Female; Glial Fibrillary Acidic Protein; Guinea Pigs; Medulla Oblongata; Neurons; Pregnancy; Prenatal Exposure Delayed Effects; Receptor, Muscarinic M2; Receptors, Muscarinic; Smoking; Substance P; Sudden Infant Death; Tyrosine 3-Monooxygenase | 2000 |