spinosyn-a has been researched along with imidacloprid* in 2 studies
2 other study(ies) available for spinosyn-a and imidacloprid
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Foliar persistence and residual activity of four insecticides of different mode of action on the predator Engytatus varians (Hemiptera: Miridae).
A greenhouse study was conducted to investigate the degradation kinetics of spinosad, flufenoxuron, dimethoate and imidacloprid in tomato (Solanum lycopersicum L.) foliage and their residual toxicity on Engytatus varians (Distant) (Hemiptera: Miridae), a predator of the tomato psyllid Bactericera cockerelli (Sulcer) (Hemiptera: Triozidae). Insecticides were sprayed at 100% and 50% of their maximum field-registered concentrations (MFRC). Starting 6 h after spraying, leaf samples were taken every 10 d for 40 d and analyzed while E. varians adults were exposed to treated leaves to evaluate residual toxicity. Immediately after application at 100% MFRC, the residue concentrations were 73.34 μg g Topics: Animals; Dimethoate; Drug Combinations; Hemiptera; Heteroptera; Insecticides; Kinetics; Macrolides; Neonicotinoids; Nitro Compounds; Plant Leaves; Solanum | 2019 |
Desensitizing and non-desensitizing subtypes of alpha-bungarotoxin-sensitive nicotinic acetylcholine receptors in cockroach neurons.
Two alpha-bungarotoxin-sensitive nicotinic receptor subtypes in cockroach neurons are identified as desensitizing (nAChD), selectively inhibitable with 100 nM imidacloprid, and non-desensitizing (nAChN), selectively inhibitable with 100 pM methyllycaconitine. Although the desensitization rate of nAChD receptors is highly variable, pharmacology is largely independent of desensitization rate. Because desensitized states tightly bind agonists, nAChD receptors are potently inhibited by neonicotinoids and specifically measured in radiolabeled imidacloprid binding assays. However, they are not usually detected in binding assays with radiolabeled alpha-bungarotoxin, which has a Kd for the resting state of 21 nM, but binds poorly to desensitized states often present in binding assays. In contrast, nAChN receptors are specifically measured in binding assays with radiolabeled alpha-bungarotoxin, which binds them with a Kd of 1.3 nM. nAChN receptors are activated by neonicotinoids at micromolar concentrations, and allosterically by spinosyn A, with an EC50 of 27 nM. Spinosyn A weakly antagonizes nAChD receptors -23% at 10 microM. The roles of the two nAChR subtypes in insecticide poisoning are discussed. Topics: Acetylcholine; Aconitine; Animals; Atropine; Bungarotoxins; Cockroaches; Dihydro-beta-Erythroidine; Electrophysiology; Guanidines; Imidazoles; Inhibitory Concentration 50; Kinetics; Macrolides; Neonicotinoids; Neurons; Nicotinic Antagonists; Nitro Compounds; Receptors, Nicotinic; Thiazoles | 2004 |