Page last updated: 2024-09-05

sb 203580 and trinitrobenzenesulfonic acid

sb 203580 has been researched along with trinitrobenzenesulfonic acid in 3 studies

Compound Research Comparison

Studies
(sb 203580)
Trials
(sb 203580)
Recent Studies (post-2010)
(sb 203580)
Studies
(trinitrobenzenesulfonic acid)
Trials
(trinitrobenzenesulfonic acid)
Recent Studies (post-2010) (trinitrobenzenesulfonic acid)
3,48941,1372,710101,042

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's3 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Drillenburg, P; Peppelenbosch, MP; Pronk, I; ten Hove, T; van den Blink, B; van Deventer, SJ1
Hollenbach, E; Malfertheiner, P; Naumann, M; Neumann, M; Roessner, A; Vieth, M1
Cabane, C; Colombel, JF; DĂ©rijard, B; Desreumaux, P; Dubuquoy, L; Gay, J; Malamut, G; Malapel, M; Tamboli, C1

Other Studies

3 other study(ies) available for sb 203580 and trinitrobenzenesulfonic acid

ArticleYear
Dichotomal role of inhibition of p38 MAPK with SB 203580 in experimental colitis.
    Gut, 2002, Volume: 50, Issue:4

    Topics: Animals; Blotting, Western; Colitis; Enzyme Activation; Enzyme Inhibitors; Enzyme-Linked Immunosorbent Assay; Imidazoles; Interferon-gamma; Mice; Mice, Inbred BALB C; Mitogen-Activated Protein Kinases; p38 Mitogen-Activated Protein Kinases; Pyridines; T-Lymphocytes; Trinitrobenzenesulfonic Acid; Tumor Necrosis Factor-alpha; Wasting Syndrome; Weight Loss

2002
Inhibition of RICK/nuclear factor-kappaB and p38 signaling attenuates the inflammatory response in a murine model of Crohn disease.
    The Journal of biological chemistry, 2005, Apr-15, Volume: 280, Issue:15

    Topics: Animals; Crohn Disease; Cytokines; Disease Models, Animal; Electrophoresis, Polyacrylamide Gel; Enzyme Activation; Enzyme Inhibitors; Female; Imidazoles; Immunoblotting; Inflammation; Mice; Mice, Inbred BALB C; Microscopy, Fluorescence; Models, Biological; NF-kappa B; p38 Mitogen-Activated Protein Kinases; Protein Kinases; Pyridines; Receptor-Interacting Protein Serine-Threonine Kinase 2; Receptor-Interacting Protein Serine-Threonine Kinases; Subcellular Fractions; Trinitrobenzenesulfonic Acid; Up-Regulation

2005
No evidence for an involvement of the p38 and JNK mitogen-activated protein in inflammatory bowel diseases.
    Digestive diseases and sciences, 2006, Volume: 51, Issue:8

    Topics: Adult; Animals; Blotting, Western; Colitis, Ulcerative; Disease Models, Animal; Enzyme Inhibitors; Female; Gene Expression; Humans; Imidazoles; Inflammatory Bowel Diseases; Interleukin-1; Intestinal Mucosa; JNK Mitogen-Activated Protein Kinases; Male; Mice; Mice, Inbred BALB C; Middle Aged; p38 Mitogen-Activated Protein Kinases; Polymerase Chain Reaction; Prognosis; Pyridines; RNA, Messenger; Trinitrobenzenesulfonic Acid; Tumor Necrosis Factor-alpha

2006