salsoline has been researched along with rasagiline in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Akao, Y; Davis, BA; Maruyama, W; Naoi, M; Youdim, MB | 1 |
| Akao, Y; Maruyama, W; Naoi, M; Yi, H | 1 |
| Akao, Y; Maruyama, W; Nakagawa, Y; Naoi, M; Shamoto-Nagai, M; Shimizu, S; Tsujimoto, Y; Yi, H; Youdim, MB | 1 |
| Akao, Y; Maruyama, W; Naoi, M; Yamaoka, Y; Yi, H | 1 |
4 other study(ies) available for salsoline and rasagiline
| Article | Year |
|---|---|
Transfection-enforced Bcl-2 overexpression and an anti-Parkinson drug, rasagiline, prevent nuclear accumulation of glyceraldehyde-3-phosphate dehydrogenase induced by an endogenous dopaminergic neurotoxin, N-methyl(R)salsolinol.
Topics: Active Transport, Cell Nucleus; Animals; Apoptosis; Cell Line; Cell Nucleus; Glyceraldehyde-3-Phosphate Dehydrogenases; Humans; Immunoblotting; Immunohistochemistry; Indans; Membrane Potentials; Mitochondria; Neuroprotective Agents; Neurotoxins; Parkinson Disease; Proto-Oncogene Proteins c-bcl-2; Salsoline Alkaloids; Signal Transduction; Tetrahydroisoquinolines; Transfection | 2001 |
Mitochondria determine the survival and death in apoptosis by an endogenous neurotoxin, N-methyl(R)salsolinol, and neuroprotection by propargylamines.
Topics: Alkynes; Animals; Apoptosis; Cell Survival; Humans; Indans; Male; Mice; Mice, Transgenic; Mitochondria; Mitochondria, Liver; Neuroprotective Agents; Proto-Oncogene Proteins c-bcl-2; Rats; Rats, Inbred Strains; Salsoline Alkaloids; Signal Transduction; Tetrahydroisoquinolines; Tumor Cells, Cultured | 2002 |
Mitochondrial permeability transition mediates apoptosis induced by N-methyl(R)salsolinol, an endogenous neurotoxin, and is inhibited by Bcl-2 and rasagiline, N-propargyl-1(R)-aminoindan.
Topics: Animals; Apoptosis; Cyclosporine; Enzyme Inhibitors; Humans; Indans; Ion Channels; Mitochondria, Liver; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Neuroblastoma; Neuroprotective Agents; Neurotoxins; Permeability; Proto-Oncogene Proteins c-bcl-2; Rats; Salsoline Alkaloids; Tetrahydroisoquinolines; Tumor Cells, Cultured | 2002 |
Involvement of type A monoamine oxidase in neurodegeneration: regulation of mitochondrial signaling leading to cell death or neuroprotection.
Topics: Cell Death; Cell Line, Tumor; Dopamine; Dose-Response Relationship, Drug; Drug Interactions; Enzyme Activation; Enzyme Inhibitors; Gene Expression Regulation; Humans; Indans; Mitochondria; Mitochondrial Membranes; Monoamine Oxidase; Nerve Degeneration; Neuroblastoma; Proto-Oncogene Proteins c-bcl-2; RNA, Small Interfering; Salsoline Alkaloids; Serotonin; Signal Transduction; Tetrahydroisoquinolines; Transfection | 2006 |