ro 31-8425 and angiotensin ii

ro 31-8425 has been researched along with angiotensin ii in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	2 (100.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Ahn, S; Barnes, WG; Lefkowitz, RJ; Wei, H	1
Bardin, S; Billet, S; Clauser, E; Conchon, S; Tacine, R	1

Other Studies

2 other study(ies) available for ro 31-8425 and angiotensin ii

Article	Year
Stable interaction between beta-arrestin 2 and angiotensin type 1A receptor is required for beta-arrestin 2-mediated activation of extracellular signal-regulated kinases 1 and 2. The Journal of biological chemistry, 2004, Nov-12, Volume: 279, Issue:46 Topics: Amino Acid Sequence; Angiotensin II; Animals; Arrestins; beta-Arrestin 2; beta-Arrestins; Cell Line; DNA-Binding Proteins; Early Growth Response Protein 1; Enzyme Activation; GTP-Binding Proteins; Humans; Immediate-Early Proteins; Indoles; Maleimides; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Molecular Sequence Data; Mutagenesis, Site-Directed; Protein Binding; Protein Kinase C; Receptor, Angiotensin, Type 1; Recombinant Fusion Proteins; RNA, Small Interfering; Second Messenger Systems; Sequence Alignment; Transcription Factors	2004
The AT1A receptor "gain-of-function" mutant N111S/delta329 is both constitutively active and hyperreactive to angiotensin II. American journal of physiology. Endocrinology and metabolism, 2006, Volume: 290, Issue:5 Topics: Angiotensin II; Angiotensin II Type 2 Receptor Blockers; Animals; Arrestins; beta-Arrestin 2; beta-Arrestins; Cell Line; Cell Proliferation; CHO Cells; Cricetinae; Cricetulus; Dose-Response Relationship, Drug; Enzyme Inhibitors; Humans; Imidazoles; Indoles; Inositol Phosphates; Maleimides; Mice; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mutation; Phosphorylation; Protein Binding; Pyridines; Receptor, Angiotensin, Type 1; Transfection	2006

Article

Year

Stable interaction between beta-arrestin 2 and angiotensin type 1A receptor is required for beta-arrestin 2-mediated activation of extracellular signal-regulated kinases 1 and 2.

The Journal of biological chemistry, 2004, Nov-12, Volume: 279, Issue:46

Topics: Amino Acid Sequence; Angiotensin II; Animals; Arrestins; beta-Arrestin 2; beta-Arrestins; Cell Line; DNA-Binding Proteins; Early Growth Response Protein 1; Enzyme Activation; GTP-Binding Proteins; Humans; Immediate-Early Proteins; Indoles; Maleimides; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Molecular Sequence Data; Mutagenesis, Site-Directed; Protein Binding; Protein Kinase C; Receptor, Angiotensin, Type 1; Recombinant Fusion Proteins; RNA, Small Interfering; Second Messenger Systems; Sequence Alignment; Transcription Factors

2004

The AT1A receptor "gain-of-function" mutant N111S/delta329 is both constitutively active and hyperreactive to angiotensin II.

American journal of physiology. Endocrinology and metabolism, 2006, Volume: 290, Issue:5

Topics: Angiotensin II; Angiotensin II Type 2 Receptor Blockers; Animals; Arrestins; beta-Arrestin 2; beta-Arrestins; Cell Line; Cell Proliferation; CHO Cells; Cricetinae; Cricetulus; Dose-Response Relationship, Drug; Enzyme Inhibitors; Humans; Imidazoles; Indoles; Inositol Phosphates; Maleimides; Mice; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Mutation; Phosphorylation; Protein Binding; Pyridines; Receptor, Angiotensin, Type 1; Transfection

2006