ro 31-8220 has been researched along with neurokinin a in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (33.33) | 18.2507 |
2000's | 2 (66.67) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Chollet, A; Nemeth, K | 1 |
Lee, K; Pinnock, RD; Preston, Z; Richardson, PJ; Widdowson, L | 1 |
Coicou, LG; Groth, RD; Mermelstein, PG; Seybold, VS | 1 |
3 other study(ies) available for ro 31-8220 and neurokinin a
Article | Year |
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A single mutation of the neurokinin-2 (NK2) receptor prevents agonist-induced desensitization. Divergent conformational requirements for NK2 receptor signaling and agonist-induced desensitization in Xenopus oocytes.
Topics: Alkaloids; Amino Acid Sequence; Animals; Base Sequence; Benzamides; Calcium; Cell Membrane; DNA Primers; Enzyme Inhibitors; Female; Humans; Ileum; In Vitro Techniques; Indoles; Kinetics; Molecular Sequence Data; Mutagenesis; Mutagenesis, Site-Directed; Neurokinin A; Oocytes; Peptide Fragments; Piperidines; Point Mutation; Protein Conformation; Protein Kinase Inhibitors; Protein Kinases; Receptors, Neurokinin-2; Recombinant Proteins; Sequence Deletion; Signal Transduction; Staurosporine; Time Factors; Xenopus | 1995 |
Tachykinins increase [3H]acetylcholine release in mouse striatum through multiple receptor subtypes.
Topics: 2-Amino-5-phosphonovalerate; Acetylcholine; Alkaloids; Animals; Benzophenanthridines; Choline O-Acetyltransferase; Corpus Striatum; Electrophysiology; Enzyme Inhibitors; Estrenes; Excitatory Amino Acid Antagonists; In Situ Hybridization; Indoles; Interneurons; Membrane Potentials; Mice; Mice, Inbred C57BL; Neurokinin A; Oligopeptides; omega-N-Methylarginine; Peptide Fragments; Phenanthridines; Phosphodiesterase Inhibitors; Piperidines; Pyrrolidinones; Quinoxalines; Quinuclidines; Receptors, Neurokinin-1; Receptors, Neurokinin-2; Receptors, Neurokinin-3; Receptors, Tachykinin; RNA, Messenger; Signal Transduction; Substance P; Tachykinins; Tetrodotoxin; Tritium | 2000 |
Substance P initiates NFAT-dependent gene expression in spinal neurons.
Topics: Animals; Animals, Newborn; Calcitonin Gene-Related Peptide; Calcium Channel Blockers; Cells, Cultured; Dose-Response Relationship, Drug; Drug Interactions; Enzyme Inhibitors; Gene Expression; Hippocampus; Indoles; Neurokinin A; Neurons, Afferent; NFATC Transcription Factors; Nimodipine; Phorbol 12,13-Dibutyrate; Quinuclidines; Rats; Rats, Sprague-Dawley; Receptors, Neurokinin-1; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Spinal Cord; Spleen; Substance P; Tacrolimus | 2006 |