resveratrol has been researched along with thromboplastin in 8 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (12.50) | 18.2507 |
| 2000's | 4 (50.00) | 29.6817 |
| 2010's | 2 (25.00) | 24.3611 |
| 2020's | 1 (12.50) | 2.80 |
| Authors | Studies |
|---|---|
| Pendurthi, UR; Rao, LV; Williams, JT | 1 |
| Mackman, N; Meng, F; Pendurthi, UR; Rao, LV | 1 |
| De Gaetano, G; Di Santo, A; Di Tommaso, R; Donati, MB; Lorenzet, R; Mezzetti, A; Napoleone, E | 1 |
| Kaur, G; Pendurthi, UR; Raul, F; Roberti, M | 1 |
| Lakota, K; Mrak-Poljsak, K; Rozman, B; Sodin-Semrl, S | 1 |
| Akhmedov, A; Breitenstein, A; Camici, GG; Elliott, PJ; Holy, EW; Lohmann, C; Lüscher, TF; Matter, CM; Spescha, R; Stein, S; Tanner, FC; Westphal, CH | 1 |
| Catalán, Ú; Fernández-Castillejo, S; Macià, A; Motilva, MJ; Solà, R | 1 |
| Abdel-Bakky, MS; Abo-Saif, AA; Abo-Youssef, AM; Darwish, MA; Messiha, BAS | 1 |
8 other study(ies) available for resveratrol and thromboplastin
| Article | Year |
|---|---|
Resveratrol, a polyphenolic compound found in wine, inhibits tissue factor expression in vascular cells : A possible mechanism for the cardiovascular benefits associated with moderate consumption of wine.
Topics: Alcohol Drinking; Cardiovascular Physiological Phenomena; Cells, Cultured; DNA; Endothelium, Vascular; Flavonoids; Humans; Phenols; Polymers; Polyphenols; Resveratrol; RNA, Messenger; Stilbenes; Thromboplastin; Transcription Factors; Transcription, Genetic; Wine | 1999 |
Mechanism of resveratrol-mediated suppression of tissue factor gene expression.
Topics: Dose-Response Relationship, Drug; Electrophoretic Mobility Shift Assay; Gene Expression Regulation; Humans; Lipopolysaccharides; Monocytes; Neoplasm Proteins; NF-kappa B; Phosphorylation; Promoter Regions, Genetic; Protein Binding; Protein Processing, Post-Translational; Resveratrol; RNA, Messenger; RNA, Neoplasm; Stilbenes; Thromboplastin; Transcription Factor AP-1; Transcription Factor RelA; Transcriptional Activation; Transfection; Tumor Cells, Cultured | 2002 |
Resveratrol and quercetin down-regulate tissue factor expression by human stimulated vascular cells.
Topics: Active Transport, Cell Nucleus; Dose-Response Relationship, Drug; Down-Regulation; Endothelium, Vascular; Humans; I-kappa B Proteins; Monocytes; Proto-Oncogene Proteins c-rel; Quercetin; Resveratrol; Stilbenes; Thromboplastin; Umbilical Veins; Wine | 2003 |
Suppression of human monocyte tissue factor induction by red wine phenolics and synthetic derivatives of resveratrol.
Topics: Cells, Cultured; Dietary Supplements; Drug Synergism; Gene Expression Regulation; Humans; Monocytes; Phenol; Quercetin; Resveratrol; Stilbenes; Structure-Activity Relationship; Thromboplastin; Wine | 2007 |
Increased responsiveness of human coronary artery endothelial cells in inflammation and coagulation.
Topics: 6-Ketoprostaglandin F1 alpha; Animals; Antioxidants; Blood Coagulation; Cells, Cultured; Coronary Vessels; Dose-Response Relationship, Drug; Endothelial Cells; Humans; Inflammation; Interleukin-1beta; Interleukin-6; Plant Extracts; Resveratrol; Stilbenes; Tea; Thromboplastin | 2009 |
Sirt1 inhibition promotes in vivo arterial thrombosis and tissue factor expression in stimulated cells.
Topics: Animals; Benzamides; Binding Sites; Cells, Cultured; Disease Models, Animal; Dose-Response Relationship, Drug; Endothelial Cells; Enzyme Activators; Genes, Reporter; Histone Deacetylase Inhibitors; Humans; Mice; Mice, Inbred C57BL; Mice, Knockout; Mitogen-Activated Protein Kinases; Naphthalenes; Naphthols; Promoter Regions, Genetic; Pyrones; Resveratrol; RNA Interference; RNA, Messenger; Sirtuin 1; Stilbenes; Thromboplastin; Thrombosis; Transcription Factor RelA; Transfection | 2011 |
Endothelial Cells Deconjugate Resveratrol Metabolites to Free Resveratrol: A Possible Role in Tissue Factor Modulation.
Topics: Cells, Cultured; Drug Stability; Endothelial Cells; Humans; Resveratrol; Thromboplastin | 2019 |
Resveratrol mitigates pancreatic TF activation and autophagy-mediated beta cell death via inhibition of CXCL16/ox-LDL pathway: A novel protective mechanism against type 1 diabetes mellitus in mice.
Topics: Animals; Antioxidants; Autophagy; Cell Death; Chemokine CXCL16; Diabetes Mellitus, Experimental; Diabetes Mellitus, Type 1; Glucose Tolerance Test; Insulin-Secreting Cells; Lipoproteins, LDL; Male; Mice; Mice, Inbred BALB C; Protective Agents; Resveratrol; Signal Transduction; Thromboplastin | 2021 |