resveratrol has been researched along with panobinostat in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (75.00) | 24.3611 |
| 2020's | 1 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Forgione, M; Hailu, GS; Mai, A; Robaa, D; Rotili, D; Sippl, W | 1 |
| Alfano, AI; Barone, S; Brindisi, M; Cassese, E; Summa, V | 1 |
| Chen, S; Dai, Y; Dent, P; Grant, S; Hock, S; Rosato, R; Yaseen, A | 1 |
| Atadja, P; Buettner, R; Cermak, JM; Jove, R; Kirschbaum, M; Kujawski, M; Scuto, A | 1 |
2 review(s) available for resveratrol and panobinostat
| Article | Year |
|---|---|
Lysine Deacetylase Inhibitors in Parasites: Past, Present, and Future Perspectives.
Topics: Animals; Antiparasitic Agents; Drug Repositioning; Helminth Proteins; Histone Deacetylase Inhibitors; Histone Deacetylases; Humans; Leishmania; Plasmodium; Protozoan Proteins; Schistosoma; Toxoplasma; Trypanosoma | 2017 |
Chasing a Breath of Fresh Air in Cystic Fibrosis (CF): Therapeutic Potential of Selective HDAC6 Inhibitors to Tackle Multiple Pathways in CF Pathophysiology.
Topics: Animals; Cystic Fibrosis; Cystic Fibrosis Transmembrane Conductance Regulator; Histone Deacetylase 6; Humans; Signal Transduction | 2022 |
2 other study(ies) available for resveratrol and panobinostat
| Article | Year |
|---|---|
Resveratrol sensitizes acute myelogenous leukemia cells to histone deacetylase inhibitors through reactive oxygen species-mediated activation of the extrinsic apoptotic pathway.
Topics: Acetylation; Apoptosis; Caspase 8; Cell Line, Tumor; DNA Damage; Drug Synergism; Histone Deacetylase Inhibitors; Humans; Hydroxamic Acids; Indoles; Leukemia, Myeloid, Acute; Metalloporphyrins; Mitochondria; NF-kappa B; Panobinostat; Reactive Oxygen Species; Receptors, TNF-Related Apoptosis-Inducing Ligand; Resveratrol; S Phase; Sirtuin 1; Stilbenes; Transcription Factor RelA; U937 Cells; Up-Regulation; Vorinostat | 2012 |
SIRT1 activation enhances HDAC inhibition-mediated upregulation of GADD45G by repressing the binding of NF-κB/STAT3 complex to its promoter in malignant lymphoid cells.
Topics: Acetylation; Apoptosis; Cell Line, Tumor; Enzyme Inhibitors; GADD45 Proteins; Heterocyclic Compounds, 4 or More Rings; Histone Deacetylase Inhibitors; Histone Deacetylases; Histones; Humans; Hydroxamic Acids; Indoles; Intracellular Signaling Peptides and Proteins; Lymphoma; NF-kappa B; Panobinostat; Phosphorylation; Promoter Regions, Genetic; Protein Binding; Proto-Oncogene Proteins c-myc; Resveratrol; RNA Interference; RNA, Small Interfering; Sirtuin 1; STAT3 Transcription Factor; Stilbenes; Up-Regulation | 2013 |