quinoxalines has been researched along with transforming growth factor alpha in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (50.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kimura, M; Ogihara, M | 1 |
| Ryu, DY; Snyderwine, EG; Thorgeirsson, SS; Weidner, V | 1 |
| Gandhi, CR; Uemura, T | 1 |
| Calvisi, DF; Conner, EA; Factor, VM; Ladu, S; Thorgeirsson, SS | 1 |
4 other study(ies) available for quinoxalines and transforming growth factor alpha
| Article | Year |
|---|---|
Stimulation by transforming growth factor-alpha of DNA synthesis and proliferation of adult rat hepatocytes in primary cultures: modulation by alpha- and beta-adrenoceptor agonists.
Topics: Adrenergic alpha-Agonists; Adrenergic Antagonists; Adrenergic beta-Agonists; Animals; Anti-Inflammatory Agents; Antibodies, Monoclonal; Brimonidine Tartrate; Cell Division; Cells, Cultured; Dexamethasone; DNA; Dose-Response Relationship, Drug; Drug Interactions; ErbB Receptors; Isoquinolines; Liver; Male; Metaproterenol; Mitogen-Activated Protein Kinase Kinases; Phenylephrine; Phosphatidylinositol 3-Kinases; Protein Kinase C; Protein Kinases; Quinoxalines; Rats; Rats, Wistar; Receptor Protein-Tyrosine Kinases; Ribosomal Protein S6 Kinases; Signal Transduction; Sphingosine; Sulfonamides; Tetradecanoylphorbol Acetate; Time Factors; Transforming Growth Factor alpha; Type C Phospholipases | 1999 |
Carcinogenicity and mutagenicity of heterocyclic amines in transgenic mouse models.
Topics: Animals; DNA, Complementary; Genes, myc; Mice; Mice, Transgenic; Mutagenesis; Mutagens; Neoplasms, Experimental; Quinolines; Quinoxalines; Transforming Growth Factor alpha | 1999 |
Inhibition of DNA synthesis in cultured hepatocytes by endotoxin-conditioned medium of activated stellate cells is transforming growth factor-beta and nitric oxide-independent.
Topics: Animals; Antibodies, Monoclonal; Cells, Cultured; Culture Media, Conditioned; DNA; Dose-Response Relationship, Drug; Endotoxins; Gene Expression Regulation, Enzymologic; Hepatocytes; Interleukin-1; Interleukin-6; Lipopolysaccharides; Liver; Male; Neutralization Tests; Nitric Oxide; Nitric Oxide Synthase; Nitric Oxide Synthase Type II; omega-N-Methylarginine; Oxadiazoles; Quinoxalines; Rats; Rats, Sprague-Dawley; RNA, Messenger; Transforming Growth Factor alpha; Transforming Growth Factor beta | 2001 |
Disruption of beta-catenin pathway or genomic instability define two distinct categories of liver cancer in transgenic mice.
Topics: alpha-Fetoproteins; Animals; beta Catenin; Cell Cycle Proteins; Cytoskeletal Proteins; Disease Models, Animal; DNA-Binding Proteins; E2F Transcription Factors; E2F1 Transcription Factor; Gene Deletion; Gene Frequency; Genes, myc; Genomic Instability; Liver Neoplasms; Loss of Heterozygosity; Mice; Mice, Transgenic; Mutation; Phenotype; Quinoxalines; Trans-Activators; Transcription Factors; Transforming Growth Factor alpha | 2004 |