Compounds > quinazolines
Page last updated: 2024-08-21

quinazolines and 2-((aminocarbonyl)amino)-5-(4-fluorophenyl)-3-thiophenecarboxamide

quinazolines has been researched along with 2-((aminocarbonyl)amino)-5-(4-fluorophenyl)-3-thiophenecarboxamide in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bai, Q; Chen, X; Du, Y; Hou, J; Nan, J; Wang, Q; Wang, Y; Yang, J; Zhang, J; Zhang, X; Zhu, N1
Bivona, TG; Chaib, I; Codony-Servat, C; Codony-Servat, J; de Los Llanos Gil, M; Karachaliou, N; Molina, MA; Ramirez, JL; Rosell, R; Solca, F1
Brands, RC; El-Mesery, M; Hartmann, S; Kübler, AC; Müller-Richter, UDA; Scheurer, MJJ; Seher, A1

Other Studies

3 other study(ies) available for quinazolines and 2-((aminocarbonyl)amino)-5-(4-fluorophenyl)-3-thiophenecarboxamide

ArticleYear
TPCA-1 is a direct dual inhibitor of STAT3 and NF-κB and regresses mutant EGFR-associated human non-small cell lung cancers.
    Molecular cancer therapeutics, 2014, Volume: 13, Issue:3

    Topics: Amides; Apoptosis; Carcinoma, Non-Small-Cell Lung; Cell Line, Tumor; Cell Proliferation; Drug Resistance, Neoplasm; Drug Synergism; ErbB Receptors; Gefitinib; Gene Expression Regulation, Neoplastic; Humans; I-kappa B Proteins; Mutation; NF-kappa B; Quinazolines; Signal Transduction; STAT3 Transcription Factor; Thiophenes

2014
Activation of signal transducer and activator of transcription 3 (STAT3) signaling in EGFR mutant non-small-cell lung cancer (NSCLC).
    Oncotarget, 2017, Jul-18, Volume: 8, Issue:29

    Topics: Afatinib; Amides; Apoptosis; Carcinoma, Non-Small-Cell Lung; Cell Line, Tumor; Cell Survival; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; ErbB Receptors; Gene Expression Regulation, Neoplastic; Humans; Lung Neoplasms; Models, Biological; Mutation; Protein Kinase Inhibitors; Quinazolines; Signal Transduction; STAT3 Transcription Factor; Thiophenes

2017
The Selection of NFκB Inhibitors to Block Inflammation and Induce Sensitisation to FasL-Induced Apoptosis in HNSCC Cell Lines Is Critical for Their Use as a Prospective Cancer Therapy.
    International journal of molecular sciences, 2019, Mar-15, Volume: 20, Issue:6

    Topics: Amides; Anti-Inflammatory Agents; Cell Line, Tumor; Cell Proliferation; Cyclopentanes; Dose-Response Relationship, Drug; Drug Screening Assays, Antitumor; Drug Synergism; Fas Ligand Protein; Head and Neck Neoplasms; Humans; Hydrocortisone; Interleukin-8; NF-kappa B; Phenyl Ethers; Pyrimidines; Quinazolines; Signal Transduction; Squamous Cell Carcinoma of Head and Neck; Thiophenes; Tumor Necrosis Factor-alpha

2019