pyrazines has been researched along with sodium arsenite in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (33.33) | 29.6817 |
2010's | 2 (66.67) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Ahsan, MH; Purchio, AF; Sambucetti, LC; West, DB; Zhang, N; Zhu, L | 1 |
Davidson, MM; Gong, X; Hei, TK; Ivanov, VN | 1 |
Gong, X; Hei, TK; Ivanov, VN | 1 |
3 other study(ies) available for pyrazines and sodium arsenite
Article | Year |
---|---|
NF-kappaB and not the MAPK signaling pathway regulates GADD45beta expression during acute inflammation.
Topics: Animals; Antigens, Differentiation; Apoptosis; Arsenites; Blotting, Northern; Blotting, Western; Boronic Acids; Bortezomib; Dexamethasone; DNA Damage; Extracellular Signal-Regulated MAP Kinases; Female; Gene Library; Genetic Vectors; I-kappa B Proteins; Inflammation; Lipopolysaccharides; Luciferases; Male; MAP Kinase Signaling System; Mice; Mice, Transgenic; NF-kappa B; NF-KappaB Inhibitor alpha; Oxidative Stress; Promoter Regions, Genetic; Proteasome Inhibitors; Protein Binding; Pyrazines; RNA, Messenger; Signal Transduction; Sodium Compounds; Thalidomide; Time Factors; Tissue Distribution; Tumor Necrosis Factor-alpha | 2005 |
Tetramethylpyrazine (TMP) protects against sodium arsenite-induced nephrotoxicity by suppressing ROS production, mitochondrial dysfunction, pro-inflammatory signaling pathways and programed cell death.
Topics: Anti-Inflammatory Agents; Antioxidants; Apoptosis; Apoptosis Regulatory Proteins; Arsenites; Autophagy; Biomarkers; Cell Line; Cytoprotection; Dose-Response Relationship, Drug; Glutathione; Humans; Inflammation Mediators; Kidney Diseases; Kidney Tubules, Proximal; Membrane Potential, Mitochondrial; Mitochondria; Oxidative Stress; Pyrazines; Reactive Oxygen Species; Signal Transduction; Sodium Compounds; Time Factors | 2015 |
2,3,5,6-Tetramethylpyrazine (TMP) down-regulated arsenic-induced heme oxygenase-1 and ARS2 expression by inhibiting Nrf2, NF-κB, AP-1 and MAPK pathways in human proximal tubular cells.
Topics: Antioxidants; Apoptosis; Arsenites; Cell Line; Cytoprotection; Dose-Response Relationship, Drug; Down-Regulation; Enzyme Inhibitors; Heme Oxygenase-1; Humans; Kidney Tubules, Proximal; Mitochondria; Mitogen-Activated Protein Kinases; NF-E2-Related Factor 2; NF-kappa B; Nuclear Proteins; Oxidative Stress; Pyrazines; Reactive Oxygen Species; Signal Transduction; Sodium Compounds; Time Factors; Transcription Factor AP-1 | 2016 |