Compounds > propranolol
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propranolol and emd 53998

propranolol has been researched along with emd 53998 in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (25.00)18.2507
2000's2 (50.00)29.6817
2010's1 (25.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chiba, S; Furukawa, Y; Hirose, M; Lakhe, M; Miyashita, Y; Nagashima, Y; Yonezawa, T1
de Zeeuw, S; Duncker, DJ; Krams, R; Trines, SA; Verdouw, PD1
Knollmann, BC; Potter, JD; Sirenko, SG1
Copeland, O; Ferenczi, MA; Marston, SB; Messer, AE; Song, W; Vikhorev, PG; Wilkinson, R1

Other Studies

4 other study(ies) available for propranolol and emd 53998

ArticleYear
Different chronotropic and inotropic effects of EMD 57033 and EMD 53998, Ca2+ sensitizers, on isolated, blood-perfused dog heart preparations.
    Japanese journal of pharmacology, 1998, Volume: 76, Issue:4

    Topics: Animals; Cardiotonic Agents; Dogs; In Vitro Techniques; Myocardial Contraction; Norepinephrine; Propranolol; Quinolines; Thiadiazines

1998
In vivo evidence that EMD 57033 restores myocardial responsiveness to intracoronary Ca(2+) in stunned myocardium.
    European journal of pharmacology, 2000, Sep-01, Volume: 403, Issue:1-2

    Topics: Animals; Calcium; Cardiotonic Agents; Coronary Vessels; Dose-Response Relationship, Drug; Female; Heart; Hemodynamics; Male; Myocardial Stunning; Propranolol; Quinolines; Stroke Volume; Swine; Thiadiazines; Vasodilator Agents; Ventricular Function, Left

2000
Differential effect of troponin T mutations on the inotropic responsiveness of mouse hearts--role of myofilament Ca2+ sensitivity increase.
    The Journal of physiology, 2006, Aug-15, Volume: 575, Issue:Pt 1

    Topics: Actin Cytoskeleton; Adrenergic beta-Antagonists; Animals; Calcium; Cardiomyopathy, Hypertrophic, Familial; Cardiotonic Agents; Exercise Tolerance; Heart; Isoproterenol; Mice; Mice, Transgenic; Mutation; Myocardial Contraction; Myocardium; Propranolol; Quinolines; Thiadiazines; Troponin T

2006
The dilated cardiomyopathy-causing mutation ACTC E361G in cardiac muscle myofibrils specifically abolishes modulation of Ca(2+) regulation by phosphorylation of troponin I.
    Biophysical journal, 2014, Nov-18, Volume: 107, Issue:10

    Topics: Actins; Animals; Calcium; Cardiomyopathy, Dilated; Carrier Proteins; Cyclic AMP-Dependent Protein Kinases; Female; Male; Mice; Mice, Transgenic; Muscle Contraction; Mutation; Myofibrils; Phosphorylation; Propranolol; Quinolines; Sarcomeres; Thiadiazines; Troponin I

2014