plicamycin has been researched along with trichostatin a in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 3 (75.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Toll, L; Waleh, N; Zaveri, NT | 1 |
Chen, TH; Chen, YT; Chiang, ER; Hung, SC; Lee, HH; Lee, YT; Liu, CL; Wang, JP; Wen, MH | 1 |
Ching, LY; Law, AY; Wong, CK; Yeung, BH | 1 |
Chiu, PT; Hsiao, G; Hsu, MJ; Hsu, YF; Huang, YH; Kuo, WH; Lin, CH; Ou, G; Sheu, JR; Yang, DS | 1 |
4 other study(ies) available for plicamycin and trichostatin a
Article | Year |
---|---|
Regulation of the prepronociceptin gene and its effect on neuronal differentiation.
Topics: Animals; Base Sequence; Bucladesine; Cell Differentiation; Cell Line; Chromatin Immunoprecipitation; Cyclic AMP Response Element-Binding Protein; Fetus; Gene Expression Regulation, Developmental; Hippocampus; Humans; Hydroxamic Acids; Isoquinolines; Mice; Molecular Sequence Data; Neurons; Nociceptin; Opioid Peptides; Plicamycin; Promoter Regions, Genetic; Protein Precursors; Rats; Rats, Sprague-Dawley; Receptors, Opioid; Sequence Alignment; Sulfonamides; Transcription, Genetic; Transfection | 2006 |
Trichostatin A inhibits TGF-β1 induced in vitro chondrogenesis of hMSCs through Sp1 suppression.
Topics: Aggrecans; Cell Differentiation; Cells, Cultured; Chondrogenesis; Collagen Type II; Histone Deacetylase Inhibitors; Humans; Hydroxamic Acids; Immunoglobulins; Mesenchymal Stem Cells; Plicamycin; Smad Proteins; SOX9 Transcription Factor; Transforming Growth Factor beta1 | 2011 |
Sp1 is a transcription repressor to stanniocalcin-1 expression in TSA-treated human colon cancer cells, HT29.
Topics: Cell Line, Tumor; Colonic Neoplasms; Enzyme Inhibitors; Gene Expression Regulation, Neoplastic; Glycoproteins; Humans; Hydroxamic Acids; Okadaic Acid; Phosphorylation; Plicamycin; Protein Synthesis Inhibitors; Repressor Proteins; Retinoblastoma Protein; Sp1 Transcription Factor; Transcription, Genetic | 2011 |
Trichostatin A and sirtinol suppressed survivin expression through AMPK and p38MAPK in HT29 colon cancer cells.
Topics: AMP-Activated Protein Kinases; Apoptosis; Benzamides; Cell Proliferation; Cell Survival; Colonic Neoplasms; Gene Expression Regulation, Neoplastic; HT29 Cells; Humans; Hydroxamic Acids; Inhibitor of Apoptosis Proteins; Luciferases; Naphthols; p38 Mitogen-Activated Protein Kinases; Plicamycin; Promoter Regions, Genetic; Protein Binding; Sp1 Transcription Factor; Survivin; Transcription Factors; Tumor Suppressor Proteins | 2012 |