pinacidil has been researched along with phosphocreatine in 7 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (14.29) | 18.2507 |
| 2000's | 5 (71.43) | 29.6817 |
| 2010's | 1 (14.29) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Cole, WC; McPherson, CD; Pierce, GN | 1 |
| Matar, W; Nosek, TM; Renaud, J; Wong, D | 1 |
| Marbán, E; O'Rourke, B; Sasaki, N; Sato, T | 1 |
| Jasmin, BJ; Lunde, JA; Matar, W; Renaud, JM | 1 |
| Liu, X; Yang, S; Ye, Y; Yu, T; Yu, Z | 1 |
| Grover, GJ; Jilkina, O; Kupriyanov, VV; Kuzio, B | 1 |
| Irie, Y; Mitsuyama, H; Mizukami, K; Tsutsui, H; Watanabe, M; Yokoshiki, H | 1 |
7 other study(ies) available for pinacidil and phosphocreatine
| Article | Year |
|---|---|
Ischemic cardioprotection by ATP-sensitive K+ channels involves high-energy phosphate preservation.
Topics: Action Potentials; Adenosine Triphosphate; Animals; Glyburide; Guanidines; Guinea Pigs; Heart; In Vitro Techniques; Membrane Potentials; Myocardial Contraction; Myocardial Ischemia; Myocardium; Phosphocreatine; Pinacidil; Potassium Channels; Reperfusion Injury; Vasodilator Agents | 1993 |
Pinacidil suppresses contractility and preserves energy but glibenclamide has no effect during muscle fatigue.
Topics: Action Potentials; Adenosine Triphosphate; Animals; Energy Metabolism; Glyburide; Hypoglycemic Agents; Mice; Mice, Inbred Strains; Muscle Contraction; Muscle Fatigue; Muscle, Skeletal; Phosphocreatine; Pinacidil; Potassium Channels; Rubidium Radioisotopes; Vasodilator Agents | 2000 |
ATP consumption by uncoupled mitochondria activates sarcolemmal K(ATP) channels in cardiac myocytes.
Topics: Adenosine Triphosphate; Animals; Atractyloside; Cells, Cultured; Dinitrofluorobenzene; Dinitrophenols; Energy Metabolism; Enzyme Inhibitors; Female; Heart; Intracellular Membranes; Kinetics; Male; Mitochondria, Heart; Myocardium; Patch-Clamp Techniques; Phosphocreatine; Pinacidil; Potassium Channels; Proton-Translocating ATPases; Rabbits; Sarcolemma; Uncoupling Agents; Vasodilator Agents | 2001 |
Denervation enhances the physiological effects of the K(ATP) channel during fatigue in EDL and soleus muscle.
Topics: Adenosine Triphosphate; Animals; Female; Gene Expression; Glyburide; Hypoglycemic Agents; Mice; Mice, Inbred Strains; Muscle Contraction; Muscle Denervation; Muscle Fatigue; Muscle, Skeletal; Phosphocreatine; Pinacidil; Potassium Channels; Potassium Channels, Inwardly Rectifying; RNA, Messenger; Rubidium Radioisotopes; Vasodilator Agents | 2001 |
Myocardial protection with pinacidil induced hyperpolarized arrest during cardiopulmonary bypass.
Topics: Adenine Nucleotides; Alkaline Phosphatase; Animals; Antihypertensive Agents; Cardiopulmonary Bypass; Creatine Kinase; Dogs; Glycogen; Heart; Heart Arrest, Induced; Hemodynamics; Hyperkalemia; L-Lactate Dehydrogenase; Lipid Peroxides; Mitochondria; Myocardial Reperfusion Injury; Myocardium; Phosphocreatine; Pinacidil; Time Factors | 2001 |
Effects of K(ATP) channel openers, P-1075, pinacidil, and diazoxide, on energetics and contractile function in isolated rat hearts.
Topics: 2,4-Dinitrophenol; Adenosine Triphosphate; Animals; Diazoxide; Guanidines; Heart; In Vitro Techniques; Magnetic Resonance Spectroscopy; Male; Myocardial Contraction; Myocardium; Oxidative Phosphorylation; Oxygen Consumption; Perfusion; Phosphocreatine; Pinacidil; Potassium Channel Blockers; Potassium Channels; Pyridines; Rats; Rats, Sprague-Dawley; Vasodilator Agents | 2002 |
Involvement of the phosphatidylinositol kinase pathway in augmentation of ATP-sensitive K(+) channel currents by hypo-osmotic stress in rat ventricular myocytes.
Topics: 1-Phosphatidylinositol 4-Kinase; Androstadienes; Animals; Calcium; Cell Size; Creatine Kinase; Dose-Response Relationship, Drug; Ion Channel Gating; KATP Channels; Male; Membrane Potentials; Membrane Transport Modulators; Myocytes, Cardiac; Osmotic Pressure; Phalloidine; Phosphocreatine; Pinacidil; Potassium; Protein Kinase Inhibitors; Rats; Rats, Inbred WKY; Signal Transduction; Time Factors; Wortmannin | 2013 |