phosphoserine has been researched along with imatinib mesylate in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 2 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Olsen, GS; Pickett, EA; Tallquist, MD | 1 |
| Kornbluth, S; Kurokawa, M; Reya, T; Zhao, C | 1 |
| Barcellos, KS; Bernusso, VA; Duarte, AS; Hansen, MD; Machado-Neto, JA; Olalla Saad, ST; Pericole, FV; Traina, F; Vieira, KP | 1 |
| Chen, YJ; Huang, HM; Huang, SY; Liu, YH; Yeh, YY | 1 |
4 other study(ies) available for phosphoserine and imatinib mesylate
| Article | Year |
|---|---|
Disruption of PDGFRalpha-initiated PI3K activation and migration of somite derivatives leads to spina bifida.
Topics: Animals; Benzamides; Cartilage; Cell Movement; Chickens; Chondrogenesis; Embryo, Mammalian; Enzyme Activation; Focal Adhesion Protein-Tyrosine Kinases; Imatinib Mesylate; Mesoderm; Mice; Mitogen-Activated Protein Kinases; Mutation; Phenotype; Phosphatidylinositol 3-Kinases; Phosphoserine; Piperazines; Proto-Oncogene Proteins c-akt; Pyrimidines; ras Proteins; Receptor, Platelet-Derived Growth Factor alpha; Signal Transduction; Somites; Spinal Dysraphism; Spine | 2008 |
Inhibition of apoptosome formation by suppression of Hsp90beta phosphorylation in tyrosine kinase-induced leukemias.
Topics: Animals; Apoptosomes; Apoptotic Protease-Activating Factor 1; Benzamides; Bone Marrow Cells; Caspase 9; Cell Line; Cytochromes c; Drug Resistance, Neoplasm; Enzyme Induction; Fusion Proteins, bcr-abl; HSP90 Heat-Shock Proteins; Humans; Imatinib Mesylate; Leukemia; Mice; Mutant Proteins; Phosphorylation; Phosphoserine; Piperazines; Protein Binding; Protein-Tyrosine Kinases; Pyrimidines | 2008 |
Imatinib restores VASP activity and its interaction with Zyxin in BCR-ABL leukemic cells.
Topics: Apoptosis; bcl-X Protein; Benzamides; Bone Marrow Cells; Cell Adhesion Molecules; Cell Proliferation; Clone Cells; Focal Adhesion Protein-Tyrosine Kinases; Fusion Proteins, bcr-abl; Gene Knockdown Techniques; Gene Silencing; Humans; Imatinib Mesylate; K562 Cells; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Microfilament Proteins; Phosphoproteins; Phosphorylation; Phosphoserine; Piperazines; Protein Binding; Pyrimidines; Zyxin | 2015 |
CD69 partially inhibits apoptosis and erythroid differentiation via CD24, and their knockdown increase imatinib sensitivity in BCR-ABL-positive cells.
Topics: Antigens, CD; Antigens, CD34; Antigens, Differentiation, T-Lymphocyte; Apoptosis; CD24 Antigen; Cell Line, Tumor; Erythroid Cells; Gene Expression Regulation, Leukemic; Humans; Imatinib Mesylate; Lectins, C-Type; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; NF-kappa B; Phosphorylation; Phosphoserine; Promoter Regions, Genetic; Signal Transduction | 2018 |