Page last updated: 2024-08-22

phenethyl isothiocyanate and imatinib mesylate

phenethyl isothiocyanate has been researched along with imatinib mesylate in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (50.00)29.6817
2010's2 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Artursson, P; Bergström, CA; Hoogstraate, J; Matsson, P; Norinder, U; Pedersen, JM1
Arlinghaus, RB; Carew, J; Giles, FJ; Huang, P; Keating, MJ; Lu, W; Trachootham, D; Zhang, H1
Balayssac, S; Ballot, C; Briand, G; Dammak, A; Dauphin, V; Formstecher, P; Idziorek, T; Jendoubi, M; Joha, S; Jonneaux, A; Kluza, J; Maboudou, P; Malet-Martino, M; Marchetti, P; Quesnel, B1
Mukherjee, A; Mukherjee, S; Roy, M; Sarkar, R1

Other Studies

4 other study(ies) available for phenethyl isothiocyanate and imatinib mesylate

ArticleYear
Prediction and identification of drug interactions with the human ATP-binding cassette transporter multidrug-resistance associated protein 2 (MRP2; ABCC2).
    Journal of medicinal chemistry, 2008, Jun-12, Volume: 51, Issue:11

    Topics: Administration, Oral; Animals; Antineoplastic Agents; Antipsychotic Agents; Antiviral Agents; ATP Binding Cassette Transporter, Subfamily B; ATP Binding Cassette Transporter, Subfamily B, Member 1; ATP Binding Cassette Transporter, Subfamily G, Member 2; ATP-Binding Cassette Transporters; Biological Transport; Cell Line; Computer Simulation; Cytochrome P-450 Enzyme System; Drug-Related Side Effects and Adverse Reactions; Estradiol; Humans; Insecta; Liver; Models, Molecular; Multidrug Resistance-Associated Protein 2; Multidrug Resistance-Associated Proteins; Neoplasm Proteins; Pharmaceutical Preparations; Pharmacology; Structure-Activity Relationship

2008
Effective killing of Gleevec-resistant CML cells with T315I mutation by a natural compound PEITC through redox-mediated mechanism.
    Leukemia, 2008, Volume: 22, Issue:6

    Topics: Aldehyde Dehydrogenase; Animals; Anticarcinogenic Agents; Antineoplastic Agents; Antioxidants; Apoptosis; Benzamides; Caspase 3; Cells, Cultured; Drug Resistance, Neoplasm; Enzyme Activation; Fusion Proteins, bcr-abl; Glutathione; Humans; Imatinib Mesylate; Immunoblotting; Isothiocyanates; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Mice; Mutation; Oxidation-Reduction; Piperazines; Precursor Cells, B-Lymphoid; Protein-Tyrosine Kinases; Pyrimidines; Reactive Oxygen Species

2008
Exploiting mitochondrial dysfunction for effective elimination of imatinib-resistant leukemic cells.
    PloS one, 2011, Volume: 6, Issue:7

    Topics: Animals; Arsenic Trioxide; Arsenicals; Benzamides; Cell Death; Cell Line, Tumor; Cell Respiration; Drug Resistance, Neoplasm; Electron Transport; Energy Metabolism; Glucose; Imatinib Mesylate; Isothiocyanates; Leukemia; Mice; Mitochondria; Models, Biological; Oxidative Stress; Oxides; Piperazines; Pyrimidines; Reactive Oxygen Species

2011
Inhibition of crosstalk between Bcr-Abl and PKC signaling by PEITC, augments imatinib sensitivity in chronic myelogenous leukemia cells.
    Chemico-biological interactions, 2015, Dec-05, Volume: 242

    Topics: Anticarcinogenic Agents; Antineoplastic Combined Chemotherapy Protocols; Cell Line, Tumor; Extracellular Signal-Regulated MAP Kinases; Fusion Proteins, bcr-abl; Humans; Imatinib Mesylate; Isothiocyanates; K562 Cells; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Lymphocytes; Molecular Targeted Therapy; Phosphorylation; Protein Kinase C; Proto-Oncogene Proteins c-raf

2015