peridinin has been researched along with butenolide* in 4 studies
4 other study(ies) available for peridinin and butenolide
| Article | Year |
|---|---|
Syntheses of ylidenbutenolide-modified derivatives of peridinin and their stereochemical and spectral characteristics.
Peridinin is a light-harvesting carotenoid found in oceanic photosynthetic organisms. It possesses a unique gamma-ylidenbutenolide function and engages in energy transfer to chlorophyll a with very high (>90%) efficiency. In order to examine the relationship between the unique structure of peridinin and its facility in carrying out energy transfer, we have synthesized two different ylidenbutenolide-modified derivatives of peridinin. In this communication, the details of the syntheses are described as are the stereochemical and spectral characteristics of the derivatives; the novel ylidenbutenolide functional group stabilizes the molecule and maintains the conjugated pi-electron system in an all-trans configuration. Topics: 4-Butyrolactone; Carotenoids; Molecular Structure; Spectrum Analysis; Stereoisomerism | 2010 |
Novel strategy for the synthesis of the butenolide moiety of peridinin.
Topics: 4-Butyrolactone; Carotenoids | 2005 |
The Stille reaction in the synthesis of carotenoid butenolides: synthesis of 6'-epi-peridinin.
A new strategy for carotenoid butenolides has been developed that is based in part in halogen-selective Stille cross-coupling of dihalogenated ylidenebutenolide segment 2 and highly functionalized alkenylstannanes. [reaction: see text] Topics: 4-Butyrolactone; Carotenoids; Models, Molecular; Molecular Conformation; Palladium | 2005 |
Inhibitory effects of (1E,3E,5E,7E)-5-hydroxy-4-(8-phenyl-1,3,5,7- octatetraenyl)-2(5H)-furanone on proliferation of human malignant tumor cells.
A butenolide compound (1E,3E,5E,7E)-5-hydroxy-4-(8-phenyl-1,3,5,7- octatetraenyl)-2(5H)-furanone (KNK-41), was shown to have strong anti-tumor activity. KNK-41 inhibited the proliferation of various kinds of human malignant tumor cells, such as HeLa (cervical carcinoma), HGC-27 (gastric cancer), PANC-1 (pancreatic cancer) and GOTO (neuroblastoma). Flow cytometric analysis indicated that KNK-41 caused an arrest in G0/G1 phase of the cell cycle. However, it scarcely affected DNA synthesis and the level of c-myc mRNA. These results suggest that the growth-inhibitory effect of KNK-41 is the result of G0/G1 arrest and not of the suppression of DNA synthesis and/or c-myc expression. Topics: 4-Butyrolactone; Antineoplastic Agents; Blotting, Northern; Carotenoids; Cell Division; DNA, Neoplasm; Flow Cytometry; Furans; Genes, myc; HeLa Cells; Humans; Neoplasm Proteins; Neuroblastoma; Pancreatic Neoplasms; RNA, Messenger; Stomach Neoplasms; Thymidine; Tumor Cells, Cultured | 1992 |