peptide elongation factor 2 has been researched along with tetradecanoylphorbol acetate in 8 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 6 (75.00) | 18.7374 |
| 1990's | 2 (25.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Gschwendt, M; Kittstein, W; Marks, F | 4 |
| Gschwendt, M; Kittstein, W; Marks, F; Mieskes, G | 1 |
| Gupta, RS; Singh, B; Stetsko, DK | 1 |
| Clark, BF; Derventzi, A; Rattan, SI | 1 |
| Chen, HC; Huang, FL; Li, H | 1 |
8 other study(ies) available for peptide elongation factor 2 and tetradecanoylphorbol acetate
| Article | Year |
|---|---|
The immunosuppressant FK-506, like cyclosporins and didemnin B, inhibits calmodulin-dependent phosphorylation of the elongation factor 2 in vitro and biological effects of the phorbol ester TPA on mouse skin in vivo.
Topics: Animals; Binding Sites; Calmodulin; Cyclosporins; Depsipeptides; Female; Immunosuppressive Agents; In Vitro Techniques; Mice; Peptide Elongation Factor 2; Peptide Elongation Factors; Peptides, Cyclic; Phosphorylation; Pyridines; Skin; Tacrolimus; Tetradecanoylphorbol Acetate | 1989 |
A type 2A protein phosphatase dephosphorylates the elongation factor 2 and is stimulated by the phorbol ester TPA in mouse epidermis in vivo.
Topics: Animals; Cytosol; Enzyme Activation; Epidermis; Ethers, Cyclic; Heparin; Mice; Molecular Weight; Okadaic Acid; Peptide Elongation Factor 2; Peptide Elongation Factors; Phosphoprotein Phosphatases; Protein Phosphatase 1; Protein Phosphatase 2; Tetradecanoylphorbol Acetate | 1989 |
Inhibition of metabolic cooperation by phorbol esters in a cell culture system based on adenosine kinase deficient mutants of V79 cells.
Topics: Adenosine; Adenosine Kinase; Animals; Cell Communication; Cell Count; Cell Line; Cricetinae; Cricetulus; Diphtheria Toxin; Dose-Response Relationship, Drug; Drug Resistance; Mutation; Peptide Elongation Factor 2; Peptide Elongation Factors; Phorbol Esters; Phorbols; Phosphorylation; Phosphotransferases; Tetradecanoylphorbol Acetate; Tubercidin | 1985 |
Ciclosporin inhibits phorbol-ester-induced hyperplastic transformation and tumor promotion in mouse skin probably by suppression of Ca2+/calmodulin-dependent processes such as phosphorylation of elongation factor 2.
Topics: Animals; Calcium; Calmodulin; Cell Transformation, Neoplastic; Chromatography, DEAE-Cellulose; Cocarcinogenesis; Cycloheximide; Cyclosporins; Cytosol; Electrophoresis, Polyacrylamide Gel; Mice; Peptide Elongation Factor 2; Peptide Elongation Factors; Phosphorus Radioisotopes; Phosphorylation; Skin; Skin Neoplasms; Tetradecanoylphorbol Acetate | 1988 |
The weak immunosuppressant cyclosporine D as well as the immunologically inactive cyclosporine H are potent inhibitors in vivo of phorbol ester TPA-induced biological effects in mouse skin and of Ca2+/calmodulin dependent EF-2 phosphorylation in vitro.
Topics: Alkaline Phosphatase; Animals; Cyclosporine; Cyclosporins; Cytosol; DNA Replication; Mice; Peptide Elongation Factor 2; Peptide Elongation Factors; Phosphorylation; Protein Biosynthesis; Protein Kinases; Skin; Structure-Activity Relationship; Tetradecanoylphorbol Acetate | 1988 |
Effect of tumor promoting phorbol ester TPA on epidermal protein synthesis: stimulation of an elongation factor 2 phosphatase activity by TPA in vivo.
Topics: Animals; Epidermis; Mice; Molecular Weight; Peptide Elongation Factor 2; Peptide Elongation Factors; Phosphorylation; Protein Biosynthesis; Skin; Tetradecanoylphorbol Acetate | 1988 |
Phorbol ester PMA stimulates protein synthesis and increases the levels of active elongation factors EF-1 alpha and EF-2 in ageing human fibroblasts.
Topics: Aging; Dose-Response Relationship, Drug; Fibroblasts; Humans; Osmolar Concentration; Peptide Elongation Factor 1; Peptide Elongation Factor 2; Peptide Elongation Factors; Protein Biosynthesis; Tetradecanoylphorbol Acetate | 1993 |
Identification of a rapidly dephosphorylating 95-kDa protein as elongation factor 2 during 8-Br-cAMP treatment of N1E115 neuroblastoma cells.
Topics: 8-Bromo Cyclic Adenosine Monophosphate; Amino Acid Sequence; Animals; Cell Differentiation; Cyclic AMP-Dependent Protein Kinases; Mice; Molecular Sequence Data; Molecular Weight; Nerve Tissue Proteins; Neuroblastoma; Neurons; Peptide Elongation Factor 2; Peptide Elongation Factors; Phosphoproteins; Phosphorylation; Protein Kinase C; Tetradecanoylphorbol Acetate; Tumor Cells, Cultured | 1995 |