pd 98059 and azacitidine

pd 98059 has been researched along with azacitidine in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	2 (100.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Bellows, DS; Clarke, ID; Diamandis, P; Dirks, PB; Graham, J; Jamieson, LG; Ling, EK; Sacher, AG; Tyers, M; Ward, RJ; Wildenhain, J	1
Der, CJ; Frantz, K; Muniz-Medina, VM; Pruitt, K; Rangnekar, VM; Rojas, RJ; Shields, JM; Ulkü, AS	1

Other Studies

2 other study(ies) available for pd 98059 and azacitidine

Article	Year
Chemical genetics reveals a complex functional ground state of neural stem cells. Nature chemical biology, 2007, Volume: 3, Issue:5 Topics: Animals; Cell Survival; Cells, Cultured; Mice; Molecular Structure; Neoplasms; Neurons; Pharmaceutical Preparations; Sensitivity and Specificity; Stem Cells	2007
Ras-mediated loss of the pro-apoptotic response protein Par-4 is mediated by DNA hypermethylation through Raf-independent and Raf-dependent signaling cascades in epithelial cells. The Journal of biological chemistry, 2005, Jun-17, Volume: 280, Issue:24 Topics: Agar; Alleles; Animals; Apoptosis; Apoptosis Regulatory Proteins; Azacitidine; Blotting, Northern; Blotting, Western; Butadienes; Cell Line; Cell Line, Tumor; Cell Transformation, Neoplastic; Decitabine; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA Methyltransferase 3A; DNA, Complementary; Down-Regulation; Enzyme Inhibitors; Epithelial Cells; Female; Fibroblasts; Flavonoids; Genetic Vectors; Humans; Intracellular Signaling Peptides and Proteins; Mice; Mutation; NIH 3T3 Cells; Nitriles; Ovary; Phosphatidylinositol 3-Kinases; Polymerase Chain Reaction; raf Kinases; Rats; RNA; RNA, Messenger; Signal Transduction; Up-Regulation	2005

Article

Year

Chemical genetics reveals a complex functional ground state of neural stem cells.

Nature chemical biology, 2007, Volume: 3, Issue:5

Topics: Animals; Cell Survival; Cells, Cultured; Mice; Molecular Structure; Neoplasms; Neurons; Pharmaceutical Preparations; Sensitivity and Specificity; Stem Cells

2007

Ras-mediated loss of the pro-apoptotic response protein Par-4 is mediated by DNA hypermethylation through Raf-independent and Raf-dependent signaling cascades in epithelial cells.

The Journal of biological chemistry, 2005, Jun-17, Volume: 280, Issue:24

Topics: Agar; Alleles; Animals; Apoptosis; Apoptosis Regulatory Proteins; Azacitidine; Blotting, Northern; Blotting, Western; Butadienes; Cell Line; Cell Line, Tumor; Cell Transformation, Neoplastic; Decitabine; DNA (Cytosine-5-)-Methyltransferase 1; DNA (Cytosine-5-)-Methyltransferases; DNA Methylation; DNA Methyltransferase 3A; DNA, Complementary; Down-Regulation; Enzyme Inhibitors; Epithelial Cells; Female; Fibroblasts; Flavonoids; Genetic Vectors; Humans; Intracellular Signaling Peptides and Proteins; Mice; Mutation; NIH 3T3 Cells; Nitriles; Ovary; Phosphatidylinositol 3-Kinases; Polymerase Chain Reaction; raf Kinases; Rats; RNA; RNA, Messenger; Signal Transduction; Up-Regulation

2005