pd 98059 and atrial natriuretic factor (4-23)nh2, de-gln(18)-de-ser(19)-de-gly(20,22)-de-leu(21)-

pd 98059 has been researched along with atrial natriuretic factor (4-23)nh2, de-gln(18)-de-ser(19)-de-gly(20,22)-de-leu(21)- in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Anand-Srivastava, MB; Hashim, S; Li, Y2

Other Studies

2 other study(ies) available for pd 98059 and atrial natriuretic factor (4-23)nh2, de-gln(18)-de-ser(19)-de-gly(20,22)-de-leu(21)-

ArticleYear
Small cytoplasmic domain peptides of natriuretic peptide receptor-C attenuate cell proliferation through Gialpha protein/MAP kinase/PI3-kinase/AKT pathways.
    American journal of physiology. Heart and circulatory physiology, 2006, Volume: 291, Issue:6

    Topics: Amino Acid Sequence; Androstadienes; Angiotensin II; Animals; Arginine Vasopressin; Atrial Natriuretic Factor; Cell Line; Cell Proliferation; Endothelin-1; Flavonoids; GTP-Binding Protein alpha Subunits, Gi-Go; Mitogen-Activated Protein Kinases; Molecular Sequence Data; Muscle, Smooth, Vascular; Peptide Fragments; Pertussis Toxin; Phosphatidylinositol 3-Kinases; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-akt; Rats; Receptors, Atrial Natriuretic Factor; Signal Transduction; Vasoconstrictor Agents; Wortmannin

2006
Intracellular peptides of natriuretic peptide receptor-C inhibit vascular hypertrophy via Gqalpha/MAP kinase signaling pathways.
    Cardiovascular research, 2006, Dec-01, Volume: 72, Issue:3

    Topics: Androstadienes; Angiotensin II; Animals; Arginine Vasopressin; Atrial Natriuretic Factor; Blotting, Western; Cell Line; Cell Proliferation; Cells, Cultured; Depression, Chemical; Dose-Response Relationship, Drug; Endothelin-1; Extracellular Signal-Regulated MAP Kinases; Flavonoids; GTP-Binding Protein alpha Subunits, Gq-G11; Leucine; MAP Kinase Signaling System; Myocytes, Smooth Muscle; Peptide Fragments; Phosphoinositide-3 Kinase Inhibitors; Phosphorylation; Proto-Oncogene Proteins c-akt; Rats; Stimulation, Chemical; Wortmannin

2006