Page last updated: 2024-08-21

oxazoles and cyclin d1

oxazoles has been researched along with cyclin d1 in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's5 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chen, R; Cheng, T; Chubb, S; Gandhi, V; Hawtin, RE; Plunkett, W1
Brautigan, DL; Edelson, JR1
Brautigan, DL; Holy, M1
Abu Aboud, O; Weiss, RH; Wettersten, HI1
Cao, R; Dan, HC; Jiang, W; Ju, L; Qian, G; Wang, G; Wang, X; Wang, Y; Wu, M; Xiao, Y1

Other Studies

5 other study(ies) available for oxazoles and cyclin d1

ArticleYear
Responses in mantle cell lymphoma cells to SNS-032 depend on the biological context of each cell line.
    Cancer research, 2010, Aug-15, Volume: 70, Issue:16

    Topics: Cell Cycle; Cell Death; Cell Growth Processes; Cell Line, Tumor; Cyclin D1; Humans; Lymphoma, Mantle-Cell; Myeloid Cell Leukemia Sequence 1 Protein; Oxazoles; Proto-Oncogene Proteins c-bcl-2; RNA, Neoplasm; RNA, Small Nuclear; Thiazoles; Transfection

2010
The Discodermia calyx toxin calyculin a enhances cyclin D1 phosphorylation and degradation, and arrests cell cycle progression in human breast cancer cells.
    Toxins, 2011, Volume: 3, Issue:1

    Topics: Animals; Antineoplastic Agents; Cell Culture Techniques; Cell Cycle Checkpoints; Cyclin D1; Dose-Response Relationship, Drug; Female; Humans; Marine Toxins; MCF-7 Cells; Oxazoles; Phosphorylation; Porifera; Protein Array Analysis; Proteolysis

2011
Calyculin A from Discodermia calyx is a dual action toxin that blocks calcium influx and inhibits protein Ser/Thr phosphatases.
    Toxins, 2012, Volume: 4, Issue:10

    Topics: Apoptosis; Blotting, Western; Calcium; Calcium Channel Blockers; Cell Division; Cell Line; Cell Proliferation; Cyclin D1; Dose-Response Relationship, Drug; Epithelial Cells; Female; Fibroblasts; Fluorescent Antibody Technique; Humans; Marine Toxins; MCF-7 Cells; Oxazoles; Phosphoprotein Phosphatases; Phosphorylation

2012
Inhibition of PPARĪ± induces cell cycle arrest and apoptosis, and synergizes with glycolysis inhibition in kidney cancer cells.
    PloS one, 2013, Volume: 8, Issue:8

    Topics: Apoptosis; Carcinoma, Renal Cell; Cell Cycle Checkpoints; Cell Line; Cell Line, Tumor; Cell Survival; Cyclin D1; Cyclin-Dependent Kinase 4; Deoxyglucose; G1 Phase; Glucose; Glycolysis; Humans; Immunoblotting; Immunohistochemistry; Kidney Neoplasms; Oxazoles; PPAR alpha; Proto-Oncogene Proteins c-myc; Resting Phase, Cell Cycle; RNA Interference; Tyrosine

2013
Simvastatin induces cell cycle arrest and inhibits proliferation of bladder cancer cells via PPARĪ³ signalling pathway.
    Scientific reports, 2016, 10-25, Volume: 6

    Topics: Anilides; Antineoplastic Agents; Apoptosis; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cyclin D1; Cyclin-Dependent Kinase 4; Cyclin-Dependent Kinase 6; Enzyme Activation; Epithelial-Mesenchymal Transition; ErbB Receptors; G1 Phase Cell Cycle Checkpoints; Glycogen Synthase Kinase 3 beta; Humans; Neoplasm Invasiveness; Oxazoles; PPAR gamma; Proto-Oncogene Proteins c-akt; Simvastatin; Tyrosine; Urinary Bladder; Urinary Bladder Neoplasms

2016