nocodazole and nutlin-3a

nocodazole has been researched along with nutlin-3a in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's4 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Apontes, P; Blagosklonny, MV; Demidenko, ZN; Leontieva, OV; Li, F1
Blagosklonny, MV1
Blagosklonny, MV; Demidenko, ZN; Gudkov, AV; Leontieva, OV1
Baer, C; Breyssens, H; Brown, NR; Caballero, OL; Cebon, J; Endicott, J; Goding, CR; Hu, Y; John, T; Jones, EY; Kessler, BM; Knapp, S; Lu, M; Lu, X; Middleton, MR; Ratnayaka, I; Salter, V; Siebold, C; Sullivan, A; Sviderskaya, EV; Zhong, S1

Other Studies

4 other study(ies) available for nocodazole and nutlin-3a

ArticleYear
Exploring long-term protection of normal human fibroblasts and epithelial cells from chemotherapy in cell culture.
    Oncotarget, 2011, Volume: 2, Issue:3

    Topics: Antineoplastic Combined Chemotherapy Protocols; Cell Cycle; Cell Line, Tumor; Cytoprotection; Epithelial Cells; Fibroblasts; G1 Phase; Humans; Imidazoles; Metformin; Nocodazole; Paclitaxel; Piperazines; Sirolimus; Tumor Suppressor Protein p53

2011
The power of chemotherapeutic engineering: arresting cell cycle and suppressing senescence to protect from mitotic inhibitors.
    Cell cycle (Georgetown, Tex.), 2011, Jul-15, Volume: 10, Issue:14

    Topics: Antineoplastic Agents; Apoptosis; Cell Cycle Checkpoints; Cell Line, Tumor; Cell Proliferation; Cellular Senescence; Cyclin-Dependent Kinase Inhibitor p21; Humans; Imidazoles; Isopropyl Thiogalactoside; Mitosis; Nocodazole; Piperazines; Sirolimus; TOR Serine-Threonine Kinases; Tumor Suppressor Protein p53

2011
Elimination of proliferating cells unmasks the shift from senescence to quiescence caused by rapamycin.
    PloS one, 2011, Volume: 6, Issue:10

    Topics: Cell Line, Tumor; Cell Proliferation; Cell Shape; Cellular Senescence; Humans; Imidazoles; Isopropyl Thiogalactoside; Nocodazole; Piperazines; Sirolimus

2011
Restoring p53 function in human melanoma cells by inhibiting MDM2 and cyclin B1/CDK1-phosphorylated nuclear iASPP.
    Cancer cell, 2013, May-13, Volume: 23, Issue:5

    Topics: Active Transport, Cell Nucleus; Animals; Antineoplastic Agents; Apoptosis; CDC2 Protein Kinase; Cell Line, Tumor; Cell Nucleus; Cell Proliferation; Cyclin B1; Dimerization; Humans; Imidazoles; Indoles; Intracellular Signaling Peptides and Proteins; M Phase Cell Cycle Checkpoints; Melanoma; Mice; Neoplasm Metastasis; Nocodazole; Phosphorylation; Piperazines; Proto-Oncogene Proteins c-mdm2; Repressor Proteins; Sulfonamides; Triazoles; Tumor Suppressor Protein p53; Vemurafenib; Xenograft Model Antitumor Assays

2013