Page last updated: 2024-08-21

nitrosobenzylmethylamine and eflornithine

nitrosobenzylmethylamine has been researched along with eflornithine in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (33.33)18.2507
2000's2 (66.67)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Fong, LY; Magee, PN; Pegg, AE1
Fong, LY; Magee, PN; Nguyen, VT; Pegg, AE1
Feith, DJ; Fong, LY; Pegg, AE1

Other Studies

3 other study(ies) available for nitrosobenzylmethylamine and eflornithine

ArticleYear
Alpha-difluoromethylornithine inhibits N-nitrosomethylbenzylamine-induced esophageal carcinogenesis in zinc-deficient rats: effects on esophageal cell proliferation and apoptosis.
    Cancer research, 1998, Dec-01, Volume: 58, Issue:23

    Topics: Animals; Anticarcinogenic Agents; Apoptosis; bcl-2-Associated X Protein; Biogenic Polyamines; Carcinogens; Cell Division; Cocarcinogenesis; Diet; Dimethylnitrosamine; Eflornithine; Esophageal Neoplasms; Esophagus; Male; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-bcl-2; Rats; Rats, Sprague-Dawley; Zinc

1998
Alpha-difluoromethylornithine induction of apoptosis: a mechanism which reverses pre-established cell proliferation and cancer initiation in esophageal carcinogenesis in zinc-deficient rats.
    Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, 2001, Volume: 10, Issue:3

    Topics: Analysis of Variance; Animals; Apoptosis; bcl-2-Associated X Protein; Blotting, Western; Cell Division; Cells, Cultured; Cyclin-Dependent Kinase Inhibitor p16; Diet; Dimethylnitrosamine; Disease Models, Animal; Drug Interactions; Eflornithine; Enzyme Inhibitors; Esophageal Neoplasms; Genes, bcl-2; Immunohistochemistry; Male; Polyamines; Proliferating Cell Nuclear Antigen; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-bcl-2; Rats; Rats, Sprague-Dawley; Sensitivity and Specificity; Zinc

2001
Antizyme overexpression in transgenic mice reduces cell proliferation, increases apoptosis, and reduces N-nitrosomethylbenzylamine-induced forestomach carcinogenesis.
    Cancer research, 2003, Jul-15, Volume: 63, Issue:14

    Topics: Animals; Apoptosis; Biogenic Polyamines; Carcinogens; Cell Division; Dimethylnitrosamine; Eflornithine; Female; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Protein Biosynthesis; Proteins; Stomach Neoplasms; Zinc

2003