nifedipine has been researched along with ha 1004 in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 1 (20.00) | 18.7374 |
1990's | 4 (80.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Glembotski, CC; Hidaka, H; Irons, CE; Sei, CA | 1 |
Brown, JH; Glembotski, CC; Irons, CE; McDonough, PM; Sei, CA; Sprenkle, AB | 1 |
Asano, T; Hidaka, H | 1 |
Crowley, MR; Fineman, JR; Soifer, SJ | 1 |
Chik, CL; Ho, AK; Karpinski, E; Li, B; Liu, QY | 1 |
5 other study(ies) available for nifedipine and ha 1004
Article | Year |
---|---|
Protein kinase C and calmodulin kinase are required for endothelin-stimulated atrial natriuretic factor secretion from primary atrial myocytes.
Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester; Animals; Atrial Natriuretic Factor; Cells, Cultured; Endothelins; Heart; Heart Atria; Isoquinolines; Kinetics; Myocardium; Nifedipine; Phorbol 12,13-Dibutyrate; Phosphorylation; Piperazines; Protein Kinase C; Protein Kinase Inhibitors; Protein Kinases; Rats; Sulfonamides | 1992 |
The alpha-adrenergic stimulation of atrial natriuretic factor expression in cardiac myocytes requires calcium influx, protein kinase C, and calmodulin-regulated pathways.
Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester; Adrenergic alpha-Agonists; Animals; Atrial Natriuretic Factor; Calcium; Calmodulin; Cells, Cultured; Gene Expression; Heart; Hydrolysis; Isoquinolines; Myocardium; Nifedipine; Phenylephrine; Phosphatidylinositols; Piperazines; Protein Kinase C; Rats; Sulfonamides | 1991 |
Intracellular Ca++ antagonist, HA1004: pharmacological properties different from those of nicardipine.
Topics: Action Potentials; Animals; Calcium Channel Blockers; Coronary Circulation; Dogs; Heart Ventricles; Hemodynamics; In Vitro Techniques; Isoquinolines; Muscle Contraction; Muscle, Smooth, Vascular; Nicardipine; Nifedipine; Renal Artery; Renal Circulation; Sulfonamides; Ventricular Function | 1985 |
HA1004, an intracellular calcium antagonist, selectively attenuates pulmonary hypertension in newborn lambs.
Topics: 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid; Animals; Animals, Newborn; Calcium Channel Blockers; Hemodynamics; Hypertension, Pulmonary; Hypoxia; Infusions, Intravenous; Isoquinolines; Muscle Relaxation; Nifedipine; Prostaglandin Endoperoxides, Synthetic; Protein Kinase Inhibitors; Pulmonary Alveoli; Regression Analysis; Sheep; Sulfonamides; Thromboxane A2 | 1994 |
cGMP inhibits L-type Ca2+ channel currents through protein phosphorylation in rat pinealocytes.
Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester; Animals; Calcium Channel Blockers; Calcium Channels; Cells, Cultured; Cyclic AMP; Cyclic GMP; Dibutyryl Cyclic GMP; Isoquinolines; Male; Nifedipine; Nitroprusside; Norepinephrine; Patch-Clamp Techniques; Phosphorylation; Pineal Gland; Piperazines; Protein Kinase Inhibitors; Rats; Rats, Sprague-Dawley; Sulfonamides; Thionucleotides | 1995 |