niacinamide has been researched along with cytochrome c-t in 18 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (5.56) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (22.22) | 29.6817 |
| 2010's | 12 (66.67) | 24.3611 |
| 2020's | 1 (5.56) | 2.80 |
| Authors | Studies |
|---|---|
| ALIVISTOTOS, SG; WILLIAMS-ASHMAN, HG | 1 |
| Chong, ZZ; Lin, SH; Maiese, K | 1 |
| Katz, E; Lioubashevski, O; Willner, I | 1 |
| Atkins, MB; Mier, JW; Panka, DJ; Wang, W | 1 |
| Chiou, JF; Jen, YM; Liu, TZ; Shiau, CY; Tai, CJ; Wang, YH | 1 |
| An, J; Chang, YJ; Chiou, JF; Huang, MT; Liu, TZ; Tai, CJ; Wang, YH; Wei, PL; Wu, CH | 1 |
| Alfieri, RR; Belletti, S; Bonelli, MA; Bottini, A; Cavazzoni, A; Dowsett, M; Evans, DB; Fox, SB; Fumarola, C; Galetti, M; Gatti, R; Generali, D; Harris, AL; La Monica, S; Martin, LA; Petronini, PG | 1 |
| Chen, N; Chen, XQ; Huang, R; Huang, Y; Zeng, H | 1 |
| Kim, MO; Lee, HY; Naseer, MI; Suh, JW; Ullah, I; Ullah, N | 1 |
| Bull, VH; Rajalingam, K; Thiede, B | 1 |
| Buettner, R; Chang, S; Hedvat, M; Jove, R; Jove, V; Liu, L; Scuto, A; Tian, Y; Van Meter, T; Wen, W; Yang, F; Yen, Y; Yip, ML | 1 |
| Cheng, H; Wang, H; Wang, K; Wen, T | 1 |
| Barnett, AJ; Brewer, GJ; Ghosh, D; LeVault, KR | 1 |
| Cabrera, R; Cao, M; Liu, C; Nelson, DR; Ogunwobi, OO; Puszyk, WM; Tian, C; Wang, T; Zhao, X | 1 |
| Chen, S; Fan, Q; Ge, J; Laties, AM; Li, A; Liao, D; Zhang, X | 1 |
| Aigner, A; Kiprianova, I; Kögel, D; Milosch, N; Mohrenz, IV; Remy, J; Seifert, V | 1 |
| Abegg, VF; Bouitbir, J; Grünig, D; Krähenbühl, S; Mingard, C; Paech, F | 1 |
| Compernolle, V; Delabie, W; Devloo, R; Feys, HB; Maes, W; Van den Hauwe, MR; Vanhoorelbeke, K | 1 |
18 other study(ies) available for niacinamide and cytochrome c-t
| Article | Year |
|---|---|
SEROTONIN-MEDIATED OXIDATION OF DIHYDRONICOTINAMIDE DERIVATIVES BY CYTOCHROME C.
Topics: Cytochromes; Cytochromes c; Electron Transport; Manganese; NAD; Niacinamide; Oxidation-Reduction; Research; Serotonin; Spectrophotometry | 1964 |
The NAD+ precursor nicotinamide governs neuronal survival during oxidative stress through protein kinase B coupled to FOXO3a and mitochondrial membrane potential.
Topics: Animals; Binding Sites; Caspase 3; Caspase 9; Caspases; Cell Survival; Cytochromes c; DNA Fragmentation; Forkhead Transcription Factors; Membrane Potentials; Mice; Mitochondria; NAD; Neurons; Niacinamide; Nuclear Proteins; Oxidative Stress; Phosphatidylinositol 3-Kinases; Phosphorylation; Protein Serine-Threonine Kinases; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; Rats; Rats, Sprague-Dawley; Transcription Factors; Transfection | 2004 |
Magnetic field effects on bioelectrocatalytic reactions of surface-confined enzyme systems: enhanced performance of biofuel cells.
Topics: Bioelectric Energy Sources; Biosensing Techniques; Catalysis; Cytochromes c; Electrochemistry; Electron Transport Complex IV; Enzymes, Immobilized; Glucose Oxidase; Gold; L-Lactate Dehydrogenase; Magnetics; Niacinamide; Oxidation-Reduction; PQQ Cofactor | 2005 |
The Raf inhibitor BAY 43-9006 (Sorafenib) induces caspase-independent apoptosis in melanoma cells.
Topics: Apoptosis; Apoptosis Regulatory Proteins; bcl-Associated Death Protein; Benzenesulfonates; Caspases; Cell Line, Tumor; Cell Nucleus; Cytochromes c; Humans; Intracellular Signaling Peptides and Proteins; Melanoma; Mitochondria; Mitochondrial Proteins; Mitogen-Activated Protein Kinases; Niacinamide; Phenylurea Compounds; Phosphorylation; Proto-Oncogene Proteins c-bcl-2; Pyridines; raf Kinases; Sorafenib | 2006 |
Sorafenib induces preferential apoptotic killing of a drug- and radio-resistant Hep G2 cells through a mitochondria-dependent oxidative stress mechanism.
Topics: Antineoplastic Agents; Apoptosis; Benzenesulfonates; Calcium; Carcinoma, Hepatocellular; Caspase 3; Caspase 7; Cell Line, Tumor; Cell Survival; Cytochromes c; Dose-Response Relationship, Drug; Enzyme Activation; Glutathione; Humans; In Situ Nick-End Labeling; Inhibitory Concentration 50; Intracellular Space; Liver Neoplasms; Microscopy, Confocal; Mitochondria; Niacinamide; Oxidative Stress; Phenylurea Compounds; Pyridines; Reactive Oxygen Species; Sorafenib | 2009 |
Glucose-regulated protein 78 is a novel contributor to acquisition of resistance to sorafenib in hepatocellular carcinoma.
Topics: Antineoplastic Agents; Apoptosis; Benzenesulfonates; Blotting, Western; Carcinoma, Hepatocellular; Caspases; Cell Cycle; Cell Proliferation; Cytochromes c; Drug Resistance, Neoplasm; Endoplasmic Reticulum Chaperone BiP; Flow Cytometry; Heat-Shock Proteins; Humans; Liver Neoplasms; Membrane Potential, Mitochondrial; Niacinamide; Phenylurea Compounds; Pyridines; RNA, Small Interfering; Sorafenib; Tumor Cells, Cultured | 2010 |
Synergistic activity of letrozole and sorafenib on breast cancer cells.
Topics: Adaptor Proteins, Signal Transducing; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Apoptosis Inducing Factor; Aromatase; Aromatase Inhibitors; Benzenesulfonates; Breast Neoplasms; Caspase 7; Caspase 9; Cell Cycle; Cell Cycle Proteins; Cell Line, Tumor; Cell Proliferation; Cyclin D1; Cytochromes c; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; Drug Synergism; Estradiol; Female; Humans; Letrozole; Mechanistic Target of Rapamycin Complex 1; Multiprotein Complexes; Niacinamide; Nitriles; Phenylurea Compounds; Phosphoproteins; Phosphorylation; Poly (ADP-Ribose) Polymerase-1; Poly(ADP-ribose) Polymerases; Protein Kinase Inhibitors; Proteins; Proto-Oncogene Proteins c-myc; Pyridines; Retinoblastoma Protein; Ribosomal Protein S6 Kinases, 70-kDa; Sorafenib; Testosterone; Time Factors; TOR Serine-Threonine Kinases; Transfection; Triazoles | 2010 |
The multikinase inhibitor sorafenib induces caspase-dependent apoptosis in PC-3 prostate cancer cells.
Topics: Apoptosis; Benzenesulfonates; Caspase 3; Caspases; Cell Line, Tumor; Cytochromes c; Extracellular Signal-Regulated MAP Kinases; Humans; Inhibitor of Apoptosis Proteins; Male; Microtubule-Associated Proteins; Mitochondria; Myeloid Cell Leukemia Sequence 1 Protein; Niacinamide; Phenylurea Compounds; Phosphorylation; Prostatic Neoplasms; Proto-Oncogene Proteins c-bcl-2; Pyridines; Sorafenib; Survivin | 2010 |
Nicotinamide inhibits alkylating agent-induced apoptotic neurodegeneration in the developing rat brain.
Topics: Alkylating Agents; Animals; Apoptosis; bcl-2-Associated X Protein; Brain; Caspase 3; Cell Survival; Cytochromes c; Cytosol; Immunohistochemistry; In Situ Nick-End Labeling; Neurodegenerative Diseases; Neurons; Niacinamide; Proto-Oncogene Proteins c-bcl-2; Rats; Rats, Sprague-Dawley; RNA, Messenger; Thiotepa; Vitamin B Complex | 2011 |
Sorafenib-induced mitochondrial complex I inactivation and cell death in human neuroblastoma cells.
Topics: Antineoplastic Agents; Apoptosis; Apoptosis Regulatory Proteins; Benzenesulfonates; Cell Line, Tumor; Cell Shape; Cytochromes c; Down-Regulation; Enzyme Activation; Humans; Membrane Potential, Mitochondrial; Mitochondria; NADH Dehydrogenase; Neuroblastoma; Niacinamide; Phenylurea Compounds; Protein Interaction Maps; Proteolysis; Proteome; Pyridines; Reactive Oxygen Species; Signal Transduction; Sorafenib; Superoxide Dismutase | 2012 |
Bortezomib induces apoptosis and growth suppression in human medulloblastoma cells, associated with inhibition of AKT and NF-ĸB signaling, and synergizes with an ERK inhibitor.
Topics: Amino Acid Chloromethyl Ketones; Antineoplastic Agents; Apoptosis; bcl-2 Homologous Antagonist-Killer Protein; bcl-Associated Death Protein; Boronic Acids; Bortezomib; Caspase 3; Caspase 9; Cerebellar Neoplasms; Cyclin-Dependent Kinase Inhibitor p21; Cyclin-Dependent Kinase Inhibitor p27; Cytochromes c; Drug Synergism; Extracellular Signal-Regulated MAP Kinases; Humans; Medulloblastoma; NF-kappa B; Niacinamide; Phenylurea Compounds; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-akt; Pyrazines; Sorafenib; Tumor Cells, Cultured | 2012 |
Different effects of histone deacetylase inhibitors nicotinamide and trichostatin A (TSA) in C17.2 neural stem cells.
Topics: Adaptor Proteins, Signal Transducing; Analysis of Variance; Animals; Apoptosis; Carrier Proteins; Cell Cycle Proteins; Cell Line; Cell Proliferation; Cell Size; Cytochromes c; Dose-Response Relationship, Drug; Eukaryotic Initiation Factors; Gene Expression Regulation; Histone Deacetylase Inhibitors; Hydroxamic Acids; Lysosomes; Mice; Neural Stem Cells; Niacinamide; p38 Mitogen-Activated Protein Kinases; Phosphoproteins; Proto-Oncogene Proteins c-bcl-2; Time Factors | 2012 |
A reversible early oxidized redox state that precedes macromolecular ROS damage in aging nontransgenic and 3xTg-AD mouse neurons.
Topics: Adenine Nucleotides; Aging; Alzheimer Disease; Amyloid beta-Protein Precursor; Animals; Calcium; Cells, Cultured; Cerebral Cortex; Chromatography, High Pressure Liquid; Cytochromes c; Disease Models, Animal; Glutathione; Hippocampus; Humans; Hydrogen Peroxide; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Mutation; NAD; Neurons; Niacinamide; Oxidants; Oxidation-Reduction; Presenilin-1; Reactive Oxygen Species; tau Proteins; Vitamin B Complex | 2012 |
OPA1 downregulation is involved in sorafenib-induced apoptosis in hepatocellular carcinoma.
Topics: Animals; Apoptosis; Carcinoma, Hepatocellular; Cell Line, Tumor; Cytochromes c; Down-Regulation; Gene Knockdown Techniques; GTP Phosphohydrolases; Humans; Liver; Liver Neoplasms; Mice; Mice, SCID; Mitochondria; Niacinamide; Phenylurea Compounds; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; raf Kinases; ras Proteins; RNA, Small Interfering; Signal Transduction; Sorafenib; Xenograft Model Antitumor Assays | 2013 |
Dynamic mobilization of PGC-1α mediates mitochondrial biogenesis for the protection of RGC-5 cells by resveratrol during serum deprivation.
Topics: Animals; Caspase 3; Cell Line; Cell Survival; Culture Media; Cytochromes c; DNA-Binding Proteins; Gene Expression Regulation; High Mobility Group Proteins; Membrane Potential, Mitochondrial; Mice; Mitochondria; Mitochondrial Turnover; Niacinamide; Nuclear Respiratory Factor 1; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha; Protein Transport; Resveratrol; Retinal Ganglion Cells; Signal Transduction; Sirtuin 1; Stilbenes; Transcription Factors | 2013 |
Sorafenib Sensitizes Glioma Cells to the BH3 Mimetic ABT-737 by Targeting MCL1 in a STAT3-Dependent Manner.
Topics: Activating Transcription Factors; Antineoplastic Agents; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Biphenyl Compounds; Cell Line, Tumor; Cell Survival; Cytochromes c; Gene Knockdown Techniques; Glioma; Humans; Myeloid Cell Leukemia Sequence 1 Protein; Niacinamide; Nitrophenols; Peptide Hydrolases; Phenylurea Compounds; Piperazines; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-bcl-2; Pyridines; Sorafenib; STAT3 Transcription Factor; Sulfonamides; Tyrphostins | 2015 |
Mechanisms of mitochondrial toxicity of the kinase inhibitors ponatinib, regorafenib and sorafenib in human hepatic HepG2 cells.
Topics: Adenosine Triphosphate; Animals; Apoptosis; Cytochromes c; Electron Transport; Hep G2 Cells; Humans; Imidazoles; Lysosomes; Membrane Potential, Mitochondrial; Mice; Mice, Inbred C57BL; Mitochondria, Liver; Mitophagy; Necrosis; Niacinamide; Phenylurea Compounds; Protein Kinase Inhibitors; Pyridazines; Pyridines; Sorafenib | 2018 |
The senotherapeutic nicotinamide riboside raises platelet nicotinamide adenine dinucleotide levels but cannot prevent storage lesion.
Topics: Apoptosis; bcl-2 Homologous Antagonist-Killer Protein; Blood Platelets; Blood Preservation; Caspase 3; Cytochromes c; Humans; NAD; Niacinamide; Platelet Aggregation; Pyridinium Compounds | 2020 |