n-methylaspartate has been researched along with myelin basic protein in 8 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (12.50) | 18.2507 |
| 2000's | 4 (50.00) | 29.6817 |
| 2010's | 3 (37.50) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Arvanitogiannis, A; Shizgal, P | 1 |
| Benarese, M; Boroni, F; Dreano, M; Garotta, G; Pizzi, M; Sarnico, I; Spano, P; Valerio, A | 1 |
| Dos Santos, CD; Gomes-Leal, W; Picanço-Diniz, CW | 1 |
| Attwell, D; Bakiri, Y; Hamilton, NB; Káradóttir, R | 1 |
| Alghisi, M; Battistin, L; Benarese, M; Boroni, F; Pizzi, M; Sarnico, I; Spano, P; Valerio, A | 1 |
| Cross, AH; Goldberg, MP; Gonzales, E; Pitt, D | 1 |
| Freire, MA; Gomes-Leal, W; Guimarães, JS; Lima, RR; Pereira, A; Picanço-Diniz, CW | 1 |
| Casson, RJ; Chidlow, G; Ebneter, A; Wood, JP | 1 |
8 other study(ies) available for n-methylaspartate and myelin basic protein
| Article | Year |
|---|---|
Early onset of demyelination after N-methyl-D-aspartate lesions of the lateral hypothalamus.
Topics: Animals; Demyelinating Diseases; Hypothalamus; Immunohistochemistry; Male; Microinjections; Myelin Basic Protein; N-Methylaspartate; Rats; Rats, Long-Evans; Time Factors | 1999 |
Prevention of neuron and oligodendrocyte degeneration by interleukin-6 (IL-6) and IL-6 receptor/IL-6 fusion protein in organotypic hippocampal slices.
Topics: Active Transport, Cell Nucleus; Animals; Antigens, CD; Astrocytes; Brain Ischemia; Cytokine Receptor gp130; DNA-Binding Proteins; Gliosis; Hippocampus; In Vitro Techniques; Interleukin-6; Membrane Glycoproteins; Myelin Basic Protein; Myelin Proteolipid Protein; N-Methylaspartate; Nerve Degeneration; Neurons; Neuroprotective Agents; Neurotoxins; Oligodendroglia; Phosphorylation; Rats; Receptors, Interleukin-6; Recombinant Fusion Proteins; RNA, Messenger; STAT1 Transcription Factor; STAT3 Transcription Factor; Trans-Activators | 2004 |
Differential patterns of inflammatory response, axonal damage and myelin impairment following excitotoxic or ischemic damage to the trigeminal spinal nucleus of adult rats.
Topics: Amyloid beta-Protein Precursor; Analysis of Variance; Animals; Axons; Brain Ischemia; Cell Count; Demyelinating Diseases; Ectodysplasins; Endothelin-1; Inflammation; Male; Myelin Basic Protein; N-Methylaspartate; Neurotoxins; Rats; Rats, Wistar; Time Factors; Trigeminal Nucleus, Spinal | 2007 |
Testing NMDA receptor block as a therapeutic strategy for reducing ischaemic damage to CNS white matter.
Topics: Action Potentials; Animals; Animals, Newborn; Brain Ischemia; Central Nervous System; Dizocilpine Maleate; Dose-Response Relationship, Drug; Drug Interactions; Excitatory Amino Acid Agonists; Excitatory Amino Acid Antagonists; In Vitro Techniques; Memantine; Myelin Basic Protein; N-Methylaspartate; Oligodendroglia; Patch-Clamp Techniques; Protein Binding; Rats; Receptors, N-Methyl-D-Aspartate | 2008 |
Targeting IKK2 by pharmacological inhibitor AS602868 prevents excitotoxic injury to neurons and oligodendrocytes.
Topics: Analysis of Variance; Animals; Animals, Newborn; Dose-Response Relationship, Drug; Electrophoretic Mobility Shift Assay; Excitatory Amino Acid Agonists; Gene Expression Regulation; Hippocampus; I-kappa B Kinase; In Vitro Techniques; Myelin Basic Protein; N-Methylaspartate; Neurons; Neuroprotective Agents; Oligodendroglia; Pyrimidines; Rats; Rats, Wistar; Time Factors | 2008 |
Dysmyelinated axons in shiverer mice are highly vulnerable to alpha-amino-3-hydroxy-5-methylisoxazole-4-propionic acid (AMPA) receptor-mediated toxicity.
Topics: alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Biomarkers; Brain; Disease Models, Animal; Excitatory Amino Acid Agonists; Female; Hereditary Central Nervous System Demyelinating Diseases; Luminescent Proteins; Mice; Mice, Inbred C57BL; Mice, Neurologic Mutants; Movement Disorders; Myelin Basic Protein; N-Methylaspartate; Nerve Degeneration; Nerve Fibers, Myelinated; Neurotoxins; Receptors, AMPA | 2010 |
Minocycline treatment reduces white matter damage after excitotoxic striatal injury.
Topics: Animals; Anti-Bacterial Agents; Antibodies, Monoclonal; Brain; Corpus Striatum; Disease Models, Animal; Macrophage Activation; Male; Microglia; Microinjections; Minocycline; Myelin Basic Protein; N-Methylaspartate; Oligodendroglia; Rats; Rats, Wistar; Time Factors | 2010 |
The optic nerve head is the site of axonal transport disruption, axonal cytoskeleton damage and putative axonal regeneration failure in a rat model of glaucoma.
Topics: Amyloid beta-Protein Precursor; Animals; Axonal Transport; Cholera Toxin; Cytoskeleton; Disease Models, Animal; Excitatory Amino Acid Agonists; Gene Expression Regulation; Glaucoma; HSP27 Heat-Shock Proteins; Myelin Basic Protein; N-Methylaspartate; Nerve Regeneration; Nerve Tissue Proteins; Optic Disk; Organic Chemicals; Rats; Retinal Ganglion Cells; RNA, Messenger; Time Factors | 2011 |