Page last updated: 2024-09-03

n-benzyladriamycin-14-valerate and cytochrome c-t

n-benzyladriamycin-14-valerate has been researched along with cytochrome c-t in 2 studies

Compound Research Comparison

Studies
(n-benzyladriamycin-14-valerate)
Trials
(n-benzyladriamycin-14-valerate)
Recent Studies (post-2010)
(n-benzyladriamycin-14-valerate)
Studies
(cytochrome c-t)
Trials
(cytochrome c-t)
Recent Studies (post-2010) (cytochrome c-t)
250311,809215,125

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Barrett, CM; Israel, M; Lothstein, L; Savranskaya, L; Sweatman, TW1
Lothstein, L; Savranskaya, L; Sweatman, TW1

Other Studies

2 other study(ies) available for n-benzyladriamycin-14-valerate and cytochrome c-t

ArticleYear
N-benzyladriamycin-14-valerate (AD 198) activates protein kinase C-delta holoenzyme to trigger mitochondrial depolarization and cytochrome c release independently of permeability transition pore opening and Ca2+ influx.
    Anti-cancer drugs, 2006, Volume: 17, Issue:5

    Topics: Amino Acid Chloromethyl Ketones; Animals; Apoptosis; Biological Transport; Calcium; Cell Line; Cytochromes c; Doxorubicin; Enzyme Activation; Granulocyte Precursor Cells; Membrane Potential, Mitochondrial; Mice; Mitochondrial Membrane Transport Proteins; Mitochondrial Membranes; Mitochondrial Permeability Transition Pore; Protein Kinase C-delta

2006
N-Benzyladriamycin-14-valerate (AD 198) cytotoxicty circumvents Bcr-Abl anti-apoptotic signaling in human leukemia cells and also potentiates imatinib cytotoxicity.
    Leukemia research, 2007, Volume: 31, Issue:8

    Topics: Antibiotics, Antineoplastic; Apoptosis; Benzamides; Cytochromes c; Doxorubicin; Drug Resistance, Neoplasm; Drug Synergism; Fusion Proteins, bcr-abl; HL-60 Cells; Humans; Imatinib Mesylate; Immunoblotting; K562 Cells; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Phosphorylation; Piperazines; Protein Kinase C; Pyrimidines; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; STAT5 Transcription Factor

2007