n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and 15-deoxyprostaglandin j2

n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide has been researched along with 15-deoxyprostaglandin j2 in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (50.00)29.6817
2010's2 (50.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
García-Bueno, B; Leza, JC; Lizasoain, I; Lorenzo, P; Madrigal, JL; Moro, MA1
Araki, E; Ishii, N; Itabe, H; Kawada, T; Kim-Mitsuyama, S; Matsumura, T; Motoshima, H; Murata, Y; Nishikawa, T; Senokuchi, T; Taketa, K; Takeya, M; Tsuruzoe, K; Yano, M1
Arai, Y; Higuchi, H; Kohjitani, A; Maeda, S; Miyawaki, T; Shimada, M; Tomoyasu, Y1
Abraki, SB; Khalaj, L; Khodagholi, F; Shaerzadeh, F1

Trials

1 trial(s) available for n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and 15-deoxyprostaglandin j2

ArticleYear
Combination of midazolam and a cyclooxygenase-2 inhibitor inhibits lipopolysaccharide-induced interleukin-6 production in human peripheral blood mononuclear cells.
    Immunopharmacology and immunotoxicology, 2012, Volume: 34, Issue:1

    Topics: Adult; Anti-Anxiety Agents; Cyclooxygenase 2 Inhibitors; Drug Synergism; Female; Humans; Interleukin-6; Leukocytes, Mononuclear; Lipopolysaccharides; Male; Midazolam; Nitrobenzenes; Prostaglandin D2; Sulfonamides

2012

Other Studies

3 other study(ies) available for n-(2-cyclohexyloxy-4-nitrophenyl)methanesulfonamide and 15-deoxyprostaglandin j2

ArticleYear
The anti-inflammatory prostaglandin 15d-PGJ2 decreases oxidative/nitrosative mediators in brain after acute stress in rats.
    Psychopharmacology, 2005, Volume: 180, Issue:3

    Topics: Animals; Cerebral Cortex; Cyclooxygenase Inhibitors; Inflammation Mediators; Male; Nitrobenzenes; Nitrosation; Oxidation-Reduction; Prostaglandin D2; Rats; Rats, Wistar; Restraint, Physical; Stress, Physiological; Sulfonamides; Time Factors

2005
Oxidized low density lipoprotein activates peroxisome proliferator-activated receptor-alpha (PPARalpha) and PPARgamma through MAPK-dependent COX-2 expression in macrophages.
    The Journal of biological chemistry, 2008, Apr-11, Volume: 283, Issue:15

    Topics: Animals; Apolipoproteins E; Atherosclerosis; Cell Line; Cyclooxygenase 2; Cyclooxygenase Inhibitors; Fatty Acids, Unsaturated; Gene Expression Regulation, Enzymologic; Humans; Lipoproteins, LDL; Macrophages, Peritoneal; MAP Kinase Signaling System; Meloxicam; Mice; Mice, Knockout; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Nitrobenzenes; p38 Mitogen-Activated Protein Kinases; PPAR alpha; PPAR gamma; Prostaglandin D2; RNA, Messenger; Sulfonamides; Thiazines; Thiazoles

2008
Simultaneous inhibition of COX-2 and activation of PPAR-γ resulted in the same level and pattern of neuroprotection as they were targeted separately.
    Journal of molecular neuroscience : MN, 2013, Volume: 49, Issue:1

    Topics: Anilides; Animals; Apoptosis; bcl-2-Associated X Protein; Cyclooxygenase 2 Inhibitors; Hydrogen Peroxide; Neurons; Neuroprotective Agents; NF-E2-Related Factor 2; Nitrobenzenes; PC12 Cells; PPAR gamma; Prostaglandin D2; Proto-Oncogene Proteins c-bcl-2; Rats; Sulfonamides

2013