melatonin and guaifenesin

melatonin has been researched along with guaifenesin in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	2 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Ahn, KS; Jeon, CM; Kim, JC; Kim, JS; Kwon, OK; Oh, SR; Park, JW; Shin, IS; Shin, NR	1
Ahn, KS; Hong, JM; Jeon, CM; Kim, JC; Kim, JS; Kwon, OK; Lee, IC; Oh, SR; Park, JW; Shin, IS; Shin, NR	1

Other Studies

2 other study(ies) available for melatonin and guaifenesin

Article	Year
Melatonin reduces airway inflammation in ovalbumin-induced asthma. Immunobiology, 2014, Volume: 219, Issue:12 Topics: Animals; Antibody Formation; Asthma; Bronchoalveolar Lavage Fluid; Cell Line; Cytokines; Disease Models, Animal; Enzyme Activation; Female; Immunoglobulin E; Inflammation; Inflammation Mediators; Matrix Metalloproteinase 9; Melatonin; Mucus; Ovalbumin; Tumor Necrosis Factor-alpha	2014
Melatonin attenuates neutrophil inflammation and mucus secretion in cigarette smoke-induced chronic obstructive pulmonary diseases via the suppression of Erk-Sp1 signaling. Journal of pineal research, 2015, Volume: 58, Issue:1 Topics: Animals; Antioxidants; Cell Line, Tumor; Disease Models, Animal; Extracellular Signal-Regulated MAP Kinases; Humans; Inflammation; Lipopolysaccharides; Male; MAP Kinase Signaling System; Melatonin; Mice; Mucus; Neutrophil Infiltration; Neutrophils; Pulmonary Disease, Chronic Obstructive; Rats; Smoking; Sp1 Transcription Factor	2015

Article

Year

Melatonin reduces airway inflammation in ovalbumin-induced asthma.

Immunobiology, 2014, Volume: 219, Issue:12

Topics: Animals; Antibody Formation; Asthma; Bronchoalveolar Lavage Fluid; Cell Line; Cytokines; Disease Models, Animal; Enzyme Activation; Female; Immunoglobulin E; Inflammation; Inflammation Mediators; Matrix Metalloproteinase 9; Melatonin; Mucus; Ovalbumin; Tumor Necrosis Factor-alpha

2014

Melatonin attenuates neutrophil inflammation and mucus secretion in cigarette smoke-induced chronic obstructive pulmonary diseases via the suppression of Erk-Sp1 signaling.

Journal of pineal research, 2015, Volume: 58, Issue:1

Topics: Animals; Antioxidants; Cell Line, Tumor; Disease Models, Animal; Extracellular Signal-Regulated MAP Kinases; Humans; Inflammation; Lipopolysaccharides; Male; MAP Kinase Signaling System; Melatonin; Mice; Mucus; Neutrophil Infiltration; Neutrophils; Pulmonary Disease, Chronic Obstructive; Rats; Smoking; Sp1 Transcription Factor

2015