Compounds > malondialdehyde
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malondialdehyde and benzyloxycarbonylvalyl-alanyl-aspartyl fluoromethyl ketone

malondialdehyde has been researched along with benzyloxycarbonylvalyl-alanyl-aspartyl fluoromethyl ketone in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's3 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Anuradha, CD; Hirano, S; Kanno, S1
Ge, H; Hou, L; Kong, X; Liu, Z; Shi, L1
Bhaskar, AS; Chaudhari, M; Jayaraj, R; Lakshmana Rao, PV1

Other Studies

3 other study(ies) available for malondialdehyde and benzyloxycarbonylvalyl-alanyl-aspartyl fluoromethyl ketone

ArticleYear
Oxidative damage to mitochondria is a preliminary step to caspase-3 activation in fluoride-induced apoptosis in HL-60 cells.
    Free radical biology & medicine, 2001, Aug-01, Volume: 31, Issue:3

    Topics: Acetylcysteine; Aldehydes; Amino Acid Chloromethyl Ketones; Antioxidants; Apoptosis; Caspase 3; Caspases; Cysteine Proteinase Inhibitors; Glutathione; HL-60 Cells; Humans; Lipid Peroxidation; Lipid Peroxides; Malondialdehyde; Mitochondria; Oxidative Stress; Sodium Fluoride

2001
Induction of apoptosis in K562/ADM cells by gamma-linolenic acid involves lipid peroxidation and activation of caspase-3.
    Chemico-biological interactions, 2006, Aug-25, Volume: 162, Issue:2

    Topics: Amino Acid Chloromethyl Ketones; Apoptosis; Butylated Hydroxytoluene; Caspase 3; Caspases; Cell Nucleus; Cell Proliferation; Cell Survival; Dose-Response Relationship, Drug; Doxorubicin; Drug Resistance, Multiple; Drug Resistance, Neoplasm; Flow Cytometry; gamma-Linolenic Acid; Humans; K562 Cells; L-Lactate Dehydrogenase; Lipid Peroxidation; Malondialdehyde; Time Factors

2006
Oxidative stress induction by T-2 toxin causes DNA damage and triggers apoptosis via caspase pathway in human cervical cancer cells.
    Toxicology, 2009, Aug-03, Volume: 262, Issue:2

    Topics: Amino Acid Chloromethyl Ketones; Apoptosis; Cell Survival; DNA Damage; DNA, Neoplasm; Female; Glutathione; HeLa Cells; Humans; Lipid Peroxidation; Malondialdehyde; Mitochondria; Neuroprotective Agents; Oxidative Stress; Signal Transduction; T-2 Toxin; Tumor Suppressor Protein p53; Uterine Cervical Neoplasms

2009