lovastatin and u 0126
lovastatin has been researched along with u 0126 in 7 studies
Research
Studies (7)
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 4 (57.14) | 29.6817 |
2010's | 3 (42.86) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors
Authors | Studies |
---|---|
Fujii, Y; Iwaki, M; Matsuoka, H; Nishida, S; Tanimori, Y; Tsubaki, M; Yanae, M | 1 |
Inoue, R; Shiraishi, T | 1 |
Goez, P; Hagelgans, A; Hempel, U; Heyne, B; Jaross, W; Menschikowski, M; Neumeister, V; Siegert, G | 1 |
Chiariello, M; Cirillo, P; De Rosa, S; Di Palma, V; Pacileo, M; Paglia, A | 1 |
Chung, HJ; Jeong, BC; Kim, IS; Kim, OS; Kim, YJ; Koh, JT; Lee, KN; Lee, SE | 1 |
Bouvier, P; Çaku, A; Corbin, F; Dubé, J; Fradet, M; Pellerin, D | 1 |
Guan, ZZ; Tan, LC; Xiao, Y; Xiu, J; Zhao, L | 1 |
Other Studies
7 other study(ies) available for lovastatin and u 0126
Article | Year |
---|---|
Mevastatin induces apoptosis in HL60 cells dependently on decrease in phosphorylated ERK.
Topics: Antineoplastic Agents; Apoptosis; Butadienes; Caspase 3; Caspases; Cell Nucleus; Extracellular Signal-Regulated MAP Kinases; HL-60 Cells; Humans; Lovastatin; Nitriles; Phosphorylation; Polyisoprenyl Phosphates; Protein Kinase Inhibitors | 2005 |
PKCalpha is involved in phorbol ester TPA-mediated stabilization of p14ARF.
Topics: Butadienes; Cell Line, Tumor; Doxycycline; Genes, p53; Humans; Leupeptins; Lovastatin; MAP Kinase Kinase Kinases; MAP Kinase Signaling System; Nitriles; Proteasome Inhibitors; Protein Kinase C; Protein Kinase C beta; Protein Kinase C-alpha; Proto-Oncogene Proteins p21(ras); RNA, Small Interfering; Staurosporine; Tetradecanoylphorbol Acetate; Tumor Suppressor Protein p14ARF | 2005 |
Statins potentiate the IFN-gamma-induced upregulation of group IIA phospholipase A2 in human aortic smooth muscle cells and HepG2 hepatoma cells.
Topics: Amides; Aorta; Atorvastatin; Bacterial Proteins; Bacterial Toxins; Butadienes; Carcinoma, Hepatocellular; CCAAT-Enhancer-Binding Proteins; Cell Line, Tumor; Drug Synergism; Flavonoids; Group II Phospholipases A2; Heptanoic Acids; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Interferon-gamma; Janus Kinase 2; Lovastatin; Myocytes, Smooth Muscle; NF-kappa B; Nitriles; Phospholipases A; Phospholipases A2; Protein-Tyrosine Kinases; Proto-Oncogene Proteins; Pyridines; Pyrroles; RNA Stability; Up-Regulation | 2005 |
Leptin stimulated C-reactive protein production by human coronary artery endothelial cells.
Topics: Butadienes; C-Reactive Protein; Carbazoles; Cells, Cultured; Coronary Vessels; Endothelial Cells; Humans; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Indoles; Leptin; Lovastatin; Maleimides; Nitriles; Protein Kinase C; Protein Kinase C beta; Protein Kinase Inhibitors; rho-Associated Kinases; rhoA GTP-Binding Protein; RNA, Messenger; Signal Transduction; Transcription, Genetic; Up-Regulation | 2009 |
Lactone form 3-hydroxy-3-methylglutaryl-coenzyme A reductase inhibitors (statins) stimulate the osteoblastic differentiation of mouse periodontal ligament cells via the ERK pathway.
Topics: Alkaline Phosphatase; Animals; Anthraquinones; Blotting, Western; Bone Morphogenetic Protein 2; Butadienes; Calcification, Physiologic; Cell Culture Techniques; Cell Differentiation; Cell Line; Cell Survival; Coloring Agents; Enzyme Inhibitors; Extracellular Signal-Regulated MAP Kinases; Hydroxymethylglutaryl-CoA Reductase Inhibitors; Integrin-Binding Sialoprotein; Lovastatin; Mice; Mitogen-Activated Protein Kinase 1; Nitriles; Osteoblasts; Osteocalcin; Periodontal Ligament; Promoter Regions, Genetic; Simvastatin; Transcriptional Activation | 2011 |
Lovastatin corrects ERK pathway hyperactivation in fragile X syndrome: potential of platelet's signaling cascades as new outcome measures in clinical trials.
Topics: Anticholesteremic Agents; Blood Platelets; Butadienes; Cell Line; Clinical Trials as Topic; Extracellular Signal-Regulated MAP Kinases; Female; Fragile X Syndrome; Humans; Lovastatin; Male; MAP Kinase Signaling System; Megakaryocytes; Nitriles; Phosphorylation; Protein Processing, Post-Translational; Proto-Oncogene Proteins c-akt; Treatment Outcome | 2016 |
Protection against the Neurotoxic Effects of β-Amyloid Peptide on Cultured Neuronal Cells by Lovastatin Involves Elevated Expression of α7 Nicotinic Acetylcholine Receptors and Activating Phosphorylation of Protein Kinases.
Topics: Aconitine; alpha7 Nicotinic Acetylcholine Receptor; Amyloid beta-Peptides; Animals; Animals, Newborn; Butadienes; Cell Death; Cell Line, Tumor; Cell Survival; Cells, Cultured; Cholesterol; Humans; Lovastatin; Malondialdehyde; MAP Kinase Signaling System; Nerve Tissue Proteins; Neurons; Neuroprotection; Neuroprotective Agents; Neurotoxins; Nitriles; Phosphorylation; Protein Kinases; Rats, Sprague-Dawley; RNA, Messenger; RNA, Small Interfering; Superoxide Dismutase | 2018 |