Page last updated: 2024-09-04

lonafarnib and okadaic acid

lonafarnib has been researched along with okadaic acid in 1 studies

Compound Research Comparison

Studies (lonafarnib)	Trials (lonafarnib)	Recent Studies (post-2010) (lonafarnib)	Studies (okadaic acid)	Trials (okadaic acid)	Recent Studies (post-2010) (okadaic acid)
224	34	90	3,865	2	658

Research

Studies (1)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (100.00)	29.6817
2010's	0 (0.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Aggarwal, BB; Khuri, FR; Takada, Y	1

Other Studies

1 other study(ies) available for lonafarnib and okadaic acid

Article	Year
Protein farnesyltransferase inhibitor (SCH 66336) abolishes NF-kappaB activation induced by various carcinogens and inflammatory stimuli leading to suppression of NF-kappaB-regulated gene expression and up-regulation of apoptosis. The Journal of biological chemistry, 2004, Jun-18, Volume: 279, Issue:25 Topics: Active Transport, Cell Nucleus; Alkyl and Aryl Transferases; Apoptosis; Blotting, Western; Carcinogens; Cell Division; Cell Line, Tumor; Cell Survival; Cytoplasm; DNA; Dose-Response Relationship, Drug; Enzyme Activation; Enzyme Inhibitors; Gene Expression Regulation, Neoplastic; Humans; Hydrogen Peroxide; I-kappa B Kinase; Immunohistochemistry; In Situ Nick-End Labeling; Inflammation; Jurkat Cells; Models, Chemical; Neovascularization, Pathologic; NF-kappa B; Okadaic Acid; Phosphorylation; Piperidines; Protein Binding; Protein Serine-Threonine Kinases; Pyridines; ras Proteins; Smoking; Time Factors; Tumor Necrosis Factor-alpha; Up-Regulation	2004

Article

Year

Protein farnesyltransferase inhibitor (SCH 66336) abolishes NF-kappaB activation induced by various carcinogens and inflammatory stimuli leading to suppression of NF-kappaB-regulated gene expression and up-regulation of apoptosis.

The Journal of biological chemistry, 2004, Jun-18, Volume: 279, Issue:25

Topics: Active Transport, Cell Nucleus; Alkyl and Aryl Transferases; Apoptosis; Blotting, Western; Carcinogens; Cell Division; Cell Line, Tumor; Cell Survival; Cytoplasm; DNA; Dose-Response Relationship, Drug; Enzyme Activation; Enzyme Inhibitors; Gene Expression Regulation, Neoplastic; Humans; Hydrogen Peroxide; I-kappa B Kinase; Immunohistochemistry; In Situ Nick-End Labeling; Inflammation; Jurkat Cells; Models, Chemical; Neovascularization, Pathologic; NF-kappa B; Okadaic Acid; Phosphorylation; Piperidines; Protein Binding; Protein Serine-Threonine Kinases; Pyridines; ras Proteins; Smoking; Time Factors; Tumor Necrosis Factor-alpha; Up-Regulation

2004