lipid-a and myrmicacin

lipid-a has been researched along with myrmicacin* in 2 studies

Other Studies

2 other study(ies) available for lipid-a and myrmicacin

ArticleYear
Bacterial medium-chain 3-hydroxy fatty acid metabolites trigger immunity in
    Science (New York, N.Y.), 2019, 04-12, Volume: 364, Issue:6436

    In plants, cell-surface immune receptors sense molecular non-self-signatures. Lipid A of Gram-negative bacterial lipopolysaccharide is considered such a non-self-signature. The receptor kinase LIPOOLIGOSACCHARIDE-SPECIFIC REDUCED ELICITATION (LORE) mediates plant immune responses to

    Topics: Acyl-Butyrolactones; Arabidopsis; Decanoic Acids; Glycolipids; Lipid A; Lipopeptides; Pseudomonas aeruginosa

2019
Unique lipid a modifications in Pseudomonas aeruginosa isolated from the airways of patients with cystic fibrosis.
    The Journal of infectious diseases, 2007, Oct-01, Volume: 196, Issue:7

    Three structural features of lipid A (addition of palmitate [C16 fatty acid], addition of aminoarabinose [positively charged amino sugar residue], and retention of 3-hydroxydecanoate [3-OH C10 fatty acid]) were determined for Pseudomonas aeruginosa isolates from patients with cystic fibrosis (CF; n=86), from the environment (n=13), and from patients with other conditions (n=14). Among P. aeruginosa CF isolates, 100% had lipid A with palmitate, 24.6% with aminoarabinose, and 33.3% retained 3-hydroxydecanoate. None of the isolates from the environment or from patients with other conditions displayed these modifications. These results indicate that unique lipid A modifications occur in clinical P. aeruginosa CF isolates.

    Topics: Arabinose; Child; Child, Preschool; Chronic Disease; Cystic Fibrosis; Decanoic Acids; Humans; Infant; Lipid A; Lung Diseases; Palmitates; Prevalence; Pseudomonas aeruginosa; Pseudomonas Infections

2007