linoleic-acid and lignoceric-acid

linoleic-acid has been researched along with lignoceric-acid* in 4 studies

Other Studies

4 other study(ies) available for linoleic-acid and lignoceric-acid

ArticleYear
A new fatty acid ester from an edible mushroom Rhizopogon luteolus.
    Natural product research, 2016, Volume: 30, Issue:20

    Phytochemical investigation of the Rhizopogon luteolus Fr. led to the isolation of one new fatty acid ester, 3-hydroxy-2,4-dimethylheptacosyl acetate (1) together with two known compounds tetracosanoic acid (2) and ergosterol (3). 1D and 2D NMR, and MS techniques were used for structural elucidation. Phenolic and fatty acid compositions were identified using HPLC-DAD and GC-MSD, respectively. Fumaric acid was the major phenolic acid, whereas linoleic, stearic and oleic acids were the most abundant fatty acids. Antioxidant and anticholinesterase activities of the extracts and compounds (1-3) were tested spectrophotometrically. Among the extracts, hexane extract showed the highest activity in all tests, particularly in β-carotene-linoleic acid assay (IC50: 16.65 ± 1.12 μg/mL). Furthermore, compound 3 exhibited higher antioxidant and anticholinesterase activities. The study indicates that R. luteolus can be used in food, cosmetic and pharmaceutical industries.

    Topics: Acetates; Agaricales; Antioxidants; beta Carotene; Cholinesterase Inhibitors; Drug Evaluation, Preclinical; Fatty Acids; Fatty Alcohols; Hexanes; Linoleic Acid; Magnetic Resonance Spectroscopy; Molecular Structure; Plant Extracts

2016
Do seed VLCFAs trigger spongy tissue formation in Alphonso mango by inducing germination?
    Journal of biosciences, 2015, Volume: 40, Issue:2

    Spongy tissue is a physiological disorder in Alphonso mango caused by the inception of germination-associated events during fruit maturation on the tree, rendering the fruit inedible. Inter-fruit competition during active fruit growth is a major contributing factor for the disorder which leads to reduced fat content in spongy tissue affected fruits. This study was, therefore, carried out to determine the possible association between seed fats and ST formation. The study of the fat content during fruit growth showed that it increased gradually from 40 percent fruit maturity. At 70 percent maturity, however, there was a sudden increase of fat content of whole fruit, leading to acute competition and resulting in differential allocation of resources among developing fruits. As a result, the seed in spongy-tissue-affected mature ripe fruit showed a marked drop in the levels of fats and the two very long chain fatty acids (VLCFAs), tetracosanoic acid and hexacosanoic acid together with an increase of linolenic acid and a fall in oleic acid contents, which are known to be key determinants for the initiation of pre-germination events in seed. Subsequently, a rise in the level of cytokinin and gibberellins in ST seed associated with a fall in abscisic acid level clearly signalled the onset of germination. Concurrently, a significant reduction in the ratio of linolenic acid/linoleic acid in pulp led to the loss of membrane integrity, cell death and the eventual formation of spongy tissue. Based on the above, it is concluded that a significant reduction in the biosynthesis of VLCFAs in seeds during fruit growth might trigger pre-germination events followed by a cascade of biochemical changes in the pulp, leading to lipid peroxidation and membrane injury in pulp culminating in ST development. Thus, this study presents crucial experimental evidence to highlight the critical role played by VLCFAs in inducing ST formation in Alphonso mango during the pre-harvest phase of fruit growth.

    Topics: Abscisic Acid; alpha-Linolenic Acid; Cytokinins; Fatty Acids; Fruit; Gas Chromatography-Mass Spectrometry; Germination; Gibberellins; Linoleic Acid; Mangifera; Oleic Acid; Plant Diseases; Plant Growth Regulators; Superoxides

2015
Plasma phospholipid fatty acids in X-linked adrenoleukodystrophy.
    Clinical chemistry, 1996, Volume: 42, Issue:3

    Blood cell and plasma lipid classes and their fatty acids were analyzed in a child with X-linked adrenoleukodystrophy. The increase in saturated fatty acids with very long chains typical of this disease occurred almost exclusively in sphingomyelin. In this lipid, the proportion of lignoceric (24:0) and hexacosanoic (26:0) acids increased while that of 18:0, 20:0, and 24:1 decreased. In the rest of the lipid classes, but especially in cholesteryl esters and triacylglycerols, the proportion of linoleate (18:2) decreased while that of oleate (18:1) increased. In glycerophospholipids, polyunsaturated fatty acids such as 20:4n-6, 22:5n-6, and 22:6n-3 were reduced while their immediate precursors, 20:3n-6, 22:4n-6, and 22:5n-3, respectively, were relatively increased, suggesting a defect in fatty acid desaturation mechanisms. Although less pronounced, a similar trend of changes was seen in the patient's mother; in both, all alterations were more marked in serum than in blood cells.

    Topics: Adrenoleukodystrophy; Child; Fatty Acids; Fatty Acids, Unsaturated; Genetic Linkage; Humans; Linoleic Acid; Linoleic Acids; Male; Oleic Acid; Oleic Acids; Phospholipids; Sphingomyelins; X Chromosome

1996
Dietary fatty acids temporarily alter liver very long-chain fatty acid composition in mice.
    The Journal of nutrition, 1992, Volume: 122, Issue:8

    To determine the influence of dietary fatty acids on tissue very long-chain fatty acid (VLFA) composition, mice were fed four diets containing 15 g fat/100 g diet derived largely from either safflower oil, peanut oil, olive oil or glycerol trioleate oil. The diets varied widely in the composition of VLFA and other fatty acids. Digestibility of total dietary VLFA ranged from 84.6% in mice fed the glycerol trioleate diet to 96.7% in those fed the safflower oil diet. After 3 mo, the saturated VLFA composition of liver total lipids and sphingomyelin was lower in animals fed the glycerol trioleate oil diet than in mice fed most other diets. Although the saturated VLFA content of the peanut oil diet was more than 15-fold greater than that of the other diets, animals fed the peanut oil diet showed little or no selective increase in liver saturated VLFA. The VLFA composition of brain was comparable in all dietary groups. After 8 mo of feeding, the liver saturated VLFA composition tended to increase and differences between groups disappeared. Liver peroxisomal beta-oxidation of lignocerate (24:0) was similar among all dietary groups. These results demonstrate that dietary fatty acids shorter than VLFA temporarily influence the saturated VLFA composition of liver.

    Topics: Animals; Dietary Fats, Unsaturated; Digestion; Fatty Acids; Kinetics; Linoleic Acid; Linoleic Acids; Liver; Male; Mice; Mice, Inbred BALB C; Microbodies; Oleic Acid; Oleic Acids; Olive Oil; Peanut Oil; Plant Oils; Safflower Oil; Triolein

1992