l 739749 has been researched along with colforsin in 1 studies
Studies (l 739749) | Trials (l 739749) | Recent Studies (post-2010) (l 739749) | Studies (colforsin) | Trials (colforsin) | Recent Studies (post-2010) (colforsin) |
---|---|---|---|---|---|
15 | 0 | 0 | 13,460 | 46 | 1,298 |
Protein | Taxonomy | l 739749 (IC50) | colforsin (IC50) |
---|---|---|---|
nuclear receptor subfamily 0 group B member 1 | Homo sapiens (human) | 1.729 | |
5-hydroxytryptamine receptor 1A | Homo sapiens (human) | 0.041 | |
Adenylate cyclase type 1 | Homo sapiens (human) | 0.0955 |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (100.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Kim, HA; Ling, B; Ratner, N | 1 |
1 other study(ies) available for l 739749 and colforsin
Article | Year |
---|---|
Nf1-deficient mouse Schwann cells are angiogenic and invasive and can be induced to hyperproliferate: reversion of some phenotypes by an inhibitor of farnesyl protein transferase.
Topics: Alkyl and Aryl Transferases; Animals; Axons; Biomarkers; Blood; Cell Division; Cells, Cultured; Colforsin; Cyclic AMP-Dependent Protein Kinases; Enzyme Activation; Enzyme Inhibitors; Glia Maturation Factor; Hyperplasia; Mice; Mice, Mutant Strains; Mutation; Myelin P0 Protein; Neoplasm Invasiveness; Neovascularization, Pathologic; Nerve Growth Factors; Nerve Tissue Proteins; Neurofibromatosis 1; Neurofibromin 1; Oligopeptides; Phenotype; Protein Prenylation; Proteins; Schwann Cells; Transferases | 1997 |