Page last updated: 2024-09-03

ki 1769 and 2-aminoadipic acid

ki 1769 has been researched along with 2-aminoadipic acid in 1 studies

*2-Aminoadipic Acid: A metabolite in the principal biochemical pathway of lysine. It antagonizes neuroexcitatory activity modulated by the glutamate receptor, N-METHYL-D-ASPARTATE; (NMDA). [MeSH]

*2-Aminoadipic Acid: A metabolite in the principal biochemical pathway of lysine. It antagonizes neuroexcitatory activity modulated by the glutamate receptor, N-METHYL-D-ASPARTATE; (NMDA). [MeSH]

Compound Research Comparison

Studies
(ki 1769)
Trials
(ki 1769)
Recent Studies (post-2010)
(ki 1769)
Studies
(2-aminoadipic acid)
Trials
(2-aminoadipic acid)
Recent Studies (post-2010) (2-aminoadipic acid)
9015464121

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Matsushita, Y; Nagai, J; Neyama, H; Tsukahara, R; Tsukahara, T; Ueda, H1

Other Studies

1 other study(ies) available for ki 1769 and 2-aminoadipic acid

ArticleYear
Involvement of lysophosphatidic acid-induced astrocyte activation underlying the maintenance of partial sciatic nerve injury-induced neuropathic pain.
    Pain, 2018, Volume: 159, Issue:11

    Topics: 2-Aminoadipic Acid; Animals; Animals, Newborn; Astrocytes; Cells, Cultured; Chemokine CXCL1; Disease Models, Animal; Dose-Response Relationship, Drug; Glial Fibrillary Acidic Protein; Lysophospholipids; Male; Mice; Mice, Inbred C57BL; Neuralgia; Pain Measurement; Pain Threshold; Pyridines; RNA, Messenger; Sciatic Neuropathy; Spinal Cord; Up-Regulation

2018