isoxazoles has been researched along with pyrazolanthrone in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 3 (75.00) | 29.6817 |
2010's | 1 (25.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Billin, AN; Bisi, J; Donahee, M; Goodwin, B; Holt, JA; Jones, SA; Kliewer, SA; Kozarsky, KF; Luo, G; Mansfield, TA; McNeill, YY; Wang, DY | 1 |
Edwards, PA; Kast-Woelbern, HR; Lusis, AJ; Shih, DM; Wong, J; Xia, YR | 1 |
Beerheide, W; Boelsterli, UA; Latchoumycandane, C; Sattabongkot, J; Seah, QM; Tan, RC | 1 |
Chen, LW; Chen, PH; Hsu, CM; Tseng, HT | 1 |
4 other study(ies) available for isoxazoles and pyrazolanthrone
Article | Year |
---|---|
Definition of a novel growth factor-dependent signal cascade for the suppression of bile acid biosynthesis.
Topics: Animals; Anthracenes; Bile Acids and Salts; Cell Line; Cells, Cultured; Chenodeoxycholic Acid; Cholesterol 7-alpha-Hydroxylase; DNA-Binding Proteins; Enzyme Repression; Fibroblast Growth Factors; Gene Expression Regulation; Hepatocytes; Humans; Isoxazoles; JNK Mitogen-Activated Protein Kinases; Mice; Mitogen-Activated Protein Kinases; Phosphorylation; Proto-Oncogene Proteins c-jun; Receptors, Cytoplasmic and Nuclear; Recombinant Proteins; Response Elements; Signal Transduction; Transcription Factors; Transfection | 2003 |
A role for FXR and human FGF-19 in the repression of paraoxonase-1 gene expression by bile acids.
Topics: Administration, Oral; Animals; Anthracenes; Aryldialkylphosphatase; Bile Acids and Salts; Cell Line, Tumor; Chenodeoxycholic Acid; Cholesterol 7-alpha-Hydroxylase; Cholesterol, Dietary; Cholic Acid; Dietary Fats; DNA-Binding Proteins; Enzyme Inhibitors; Female; Fibroblast Growth Factors; Gene Expression; Humans; Isoxazoles; JNK Mitogen-Activated Protein Kinases; Mice; Mice, Inbred C57BL; Mice, Knockout; Mutation; Phospholipid Transfer Proteins; Promoter Regions, Genetic; Receptors, Cytoplasmic and Nuclear; Sterol Regulatory Element Binding Protein 1; Transcription Factors | 2006 |
Leflunomide or A77 1726 protect from acetaminophen-induced cell injury through inhibition of JNK-mediated mitochondrial permeability transition in immortalized human hepatocytes.
Topics: Acetaminophen; Aniline Compounds; Anthracenes; Antirheumatic Agents; Caspase 3; Cell Death; Cell Line; Crotonates; Cytochromes c; Dose-Response Relationship, Drug; Enzyme Activation; Glutathione; Hepatocytes; Humans; Hydroxybutyrates; Isoxazoles; JNK Mitogen-Activated Protein Kinases; Leflunomide; Mitochondria, Liver; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Mitogen-Activated Protein Kinase 8; Mitogen-Activated Protein Kinase 9; Nitriles; Phosphorylation; Protein Carbonylation; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-bcl-2; Toluidines | 2006 |
Peritonitis-induced peroxynitrite and lung damage depends on c-Jun NH2-terminal kinase signaling of hematopoietic cells.
Topics: Animals; Anthracenes; Blotting, Western; Dimethyl Sulfoxide; Disease Models, Animal; Hematopoiesis; Isoxazoles; JNK Mitogen-Activated Protein Kinases; Leflunomide; Lung Injury; Male; Mice; Mice, Inbred C57BL; Myeloid Cells; NF-kappa B; Nitric Oxide Synthase; Peritonitis; Peroxynitrous Acid; Rhodamines; Signal Transduction; Transcription Factor AP-1 | 2010 |