isospaglumic acid has been researched along with urea in 3 studies
| Studies (isospaglumic acid) | Trials (isospaglumic acid) | Recent Studies (post-2010) (isospaglumic acid) | Studies (urea) | Trials (urea) | Recent Studies (post-2010) (urea) |
|---|---|---|---|---|---|
| 469 | 31 | 116 | 45,450 | 2,047 | 9,188 |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kozikowski, A; Neale, JH; Yamamoto, T; Zhou, J | 1 |
| Adedoyin, MO; Neale, JH; Vicini, S | 1 |
| Borst, P; de Haas, M; Jansen, RS; Mahakena, S; van de Wetering, K | 1 |
3 other study(ies) available for isospaglumic acid and urea
| Article | Year |
|---|---|
Intracerebroventricular administration of N-acetylaspartylglutamate (NAAG) peptidase inhibitors is analgesic in inflammatory pain.
Topics: Analgesics; Animals; Dipeptides; Glutamate Carboxypeptidase II; Inflammation Mediators; Injections, Intraventricular; Male; Organophosphorus Compounds; Pain; Protease Inhibitors; Rats; Rats, Sprague-Dawley; Urea | 2008 |
Endogenous N-acetylaspartylglutamate (NAAG) inhibits synaptic plasticity/transmission in the amygdala in a mouse inflammatory pain model.
Topics: Amygdala; Animals; Behavior, Animal; Dipeptides; Disease Models, Animal; Excitatory Postsynaptic Potentials; Formaldehyde; Glutamate Carboxypeptidase II; Hyperalgesia; In Vitro Techniques; Inflammation; Mice; Models, Biological; Neuronal Plasticity; Nociceptors; Pain; Receptors, AMPA; Receptors, Metabotropic Glutamate; Receptors, N-Methyl-D-Aspartate; Synaptic Transmission; Urea | 2010 |
ATP-binding Cassette Subfamily C Member 5 (ABCC5) Functions as an Efflux Transporter of Glutamate Conjugates and Analogs.
Topics: Animals; Brain; Dipeptides; Humans; Kainic Acid; Mice; Mice, Knockout; Multidrug Resistance-Associated Proteins; Nerve Tissue Proteins; Urea | 2015 |